1.A Case of Thin Basement Membrane Nephropathy.
Byung Soo CHO ; Chang Il AHN ; Hyun Soon LEE
Journal of the Korean Pediatric Society 1987;30(3):305-308
No abstract available.
Basement Membrane*
2.Thin glomerular basement membrane disease-2 cases.
Jeong Hyun PARK ; Ji Soo PYO ; Sung Cheul OK ; Hwan Tae KIM ; In Hee LEE ; Yeong Hoon KIM ; Jong Eun JOO
Korean Journal of Nephrology 1993;12(2):165-171
No abstract available.
Glomerular Basement Membrane*
3.A Case of Anti-Glomerular Basement Membrane Gomerulonephritis Superimposed on Preexisting Membranous Nephropathy.
Korean Journal of Nephrology 2005;24(5):838-844
No abstract available.
Basement Membrane*
;
Glomerulonephritis, Membranous*
4.Anionic sites in rat alveolar basement membrane.
Ho Gak KIM ; Soo Il KIM ; Kyung Ran PARK ; Seung Moo NO ; Won Sik KIM
Korean Journal of Anatomy 1992;25(2):128-138
No abstract available.
Animals
;
Basement Membrane*
;
Rats*
5.Ultrastructural study of basement membrane of choleastoma epithelium.
Kyung You PARK ; Joong Saeng CHO ; Chang Il CHA
Korean Journal of Otolaryngology - Head and Neck Surgery 1991;34(4):649-658
No abstract available.
Basement Membrane*
;
Epithelium*
6.Basement Membrane Status Is Intact in Urticarial Dermatitis vs. Adult-Onset Atopic Dermatitis.
Tae In KIM ; Hyung Jin PARK ; Yong Yon WON ; Hyeongwon CHOI ; Ki Heon JEONG ; Ji Youn SUNG ; Min Kyung SHIN
Annals of Dermatology 2018;30(2):258-261
No abstract available.
Basement Membrane*
;
Dermatitis*
;
Dermatitis, Atopic*
7.IgA nephropathy combined with thin basement membrane nephropathy in children.
Kidney Research and Clinical Practice 2013;32(4):194-195
No abstract available.
Basement Membrane*
;
Child*
;
Glomerulonephritis, IGA*
;
Humans
;
Immunoglobulin A*
8.Ultrastructural and Immunofluorescent Features of Lymphatic Disorders in Conjunctiva.
Jung Hee JOO ; Myung Kyoo KO ; Moon Hyang PARK
Journal of the Korean Ophthalmological Society 1987;28(3):545-550
We have experienced one case of lymphangioma and one case of lymphangiectasia which was diagnosed with the histopathologic studies, including immunofluorescent and electron microscopic methods. The distribution of Ig G and Ig A within the stroma was shown, particularly in the inflammatory cells. The endothelial lined lymphatic vessel consisted of interrupted basal lamina and absence of pericyte on electron microscope.
Basement Membrane
;
Conjunctiva*
;
Lymphangioma
;
Lymphatic Vessels
;
Pericytes
9.Electron microscopic studies of epithelial adhesion complex of keratoconus.
Myeong Gyu PARK ; Kayoung YI ; Myung Kyoo KO
Journal of the Korean Ophthalmological Society 2001;42(10):1476-1482
PURPOSE: Keratoconus is a bilateral noninflammatory ecstatic disease of cornea. Clinical manifestations and treatments are well-described , but the exact pathophysiology has many debates. There are many reports on pathologic abnormalities of keratoconus, but few reports on epithelial adhesion complex. The authors investigated the abnormalities in epithelial adhesion complex of keratoconus. METHODS: Using 4 corneas from 4 recipients of penetrating keratoplasty, examination was done with transmission electron microscope (Hitachi-600, Japan) after proper fixation and staining. Central and peripheral portion of each corneal tissues were examined. RESULTS: In two tissues, severe degeneration of basement membrane and Bowman's layer were found. Some degree of abnormalities was found in other tissues, which had minimal change. Some of hemidesmosomes, the most distinct part of adhesion complex, were found only in well-maintained tissue but the distribution was abnormal. CONCLUSIONS: The fact that basal plasma membrane had selectively more degenerations and changes than intercellular plasma membrane implies pathophysiology of keratoconus on adhesion complex, basal plasma membrane, basement membrane and Bowman's layer. Further study on this issue will reveal more information as to its pathophysiology.
Basement Membrane
;
Cell Membrane
;
Cornea
;
Hemidesmosomes
;
Keratoconus*
;
Keratoplasty, Penetrating
10.Immunohistochemical Localization of Extracellular Matrix Components in Diabetic Nephropathy.
Seung Sam PAIK ; Moon Hyang PARK
Korean Journal of Pathology 1997;31(5):427-435
Normal human glomerular basement membrane (GBM) and mesangial matrix (MM) contain several different basement membrane components in varying degrees. The characteristic morphological and ultrastructural changes in patients with diabetic nephropathy are the thickening of the GBM and the expansion of the MM. In order to investigate the changes of extracellular matrix components in diabetes, the immunohistochemical localization was performed in 17 cases with different degrees using antisera to human collagen types I, III, IV, VI, fibronectin, and laminin. The following results were obtained: 1. The reactivity for collagen IV was increased in expanded MM in the diffuse glomerulosclerosis (GS). With the progression to the nodule formation, collagen IV was prominently decreased in the peripheral area of the nodules. 2. Collagen VI was increased in GBM and MM in the diffuse GS, it was especially prominent in the expanded MM. With the progression to nodule formation, collagen VI was prominently increased in the periphery of the nodules. 3. Interstitial collagen I and III were not stained in many of the cases with the diffuse GS. With the progression to nodule formation, these were slightly expressed. A lamellar pattern of positive reaction was noted at the periphery of the late nodular lesions. 4. Fibronectin was increased in GBM & MM in the diffuse GS, it was especially intense in the MM. With the progression to the nodule formation, the reactivity of antibody to the fibronectin was decreased. 5. Laminin was weakly stained along the GBM & trace in the MM, but was not changed in the nodular GS. In summary, the expanded mesangial matrix in the diffuse GS showed a markedly increased staining for collagen IV, fibronectin and collagen VI. Less intense linear staining for collagen VI, fibronectin, laminin, collagen IV and collagen III was noted along the GBM. In the nodular GS, the composition of the early nodules resembled that of the diffuse GS. However, the late nodular lesion of the nodular GS revealed decreased reactivity for collagen IV and fibronectin at the periphery of the nodule, where collagen VI and interstitial collagen I and III were increased in laminated pattern.
Basement Membrane
;
Collagen
;
Diabetic Nephropathies*
;
Extracellular Matrix*
;
Fibronectins
;
Glomerular Basement Membrane
;
Humans
;
Immune Sera
;
Laminin