1.Percutaneous Transluminal Coronary Angioplasty, PTCA.
Korean Circulation Journal 1992;22(6):905-911
No abstract available.
Angioplasty, Balloon, Coronary*
2.Emergency surgery after failed percutaneous transluminal coronary angioplasty.
Young Hwan PARK ; Hwan Kyu ROH ; Byung Chul CHANG ; Meyun Shick KANG ; Bum Koo CHO ; Sung Nok HONG ; Pill Whoon HONG
The Korean Journal of Thoracic and Cardiovascular Surgery 1992;25(1):73-78
No abstract available.
Angioplasty, Balloon, Coronary*
;
Emergencies*
3.Coronary Irradiation for the Prevention of Restenosis after Percutaneous Transluminal Coronary Angioplasty.
Korean Circulation Journal 1998;28(2):161-163
No abstract available.
Angioplasty, Balloon, Coronary*
7.Two Cases of Coronary Pseudo-Lesion Induced in the Left Circumflex Artery and the Right Coronary Artery by the Angioplasty Guide-Wire.
Myoung Seok KIM ; Chong Jin KIM ; Su Beom HEO ; Eun Ju CHO ; Jae Han PARK ; Gueng Sung CHOI ; Keon Woong MOON ; Hee Yeol KIM ; Doo Soo JEON ; Tai Ho ROH ; Jae Hyung KIM ; Kyu Bo CHOI ; Soon Jo HONG
Korean Circulation Journal 2004;34(8):799-803
Coronary pseudo-lesion is an artificial lesion that occurs during percutaneous transluminal coronary angioplasty by an angioplasty guide wire and/or a balloon as a result of a straightening of the vessel curvature. A specific treatment is not required and the condition is completely resolved after removing the angioplasty wire. There are few reports about a pseudo-lesion, particularly in the left circumflex artery. We report two cases of a coronary pseudo-lesion induced by an angioplasty guide wire; one case affecting the left circumflex artery and the other affecting the right coronary artery.
Angioplasty*
;
Angioplasty, Balloon, Coronary
;
Arteries*
;
Coronary Vessels*
8.Effectiveness of the Hugging Balloon Technique in Coronary Angioplasty for a Heavy, Encircling, Calcified Coronary Lesion.
Min Soo AHN ; Junghan YOON ; Jun Won LEE ; Kyoung Hoon LEE ; Jang Young KIM ; Byung Su YOO ; Seung Hwan LEE ; Kyung Hoon CHOE
Korean Circulation Journal 2009;39(11):499-501
We report our experience in coronary angioplasty and intravascular ultrasonography (IVUS) on a heavy, encircling, calcified lesion that was not dilated with the use of a cutting balloon and a non-compliant balloon. The angioplasty was successfully performed with a simple and inexpensive hugging balloon technique.
Angioplasty
;
Angioplasty, Balloon
;
Coronary Stenosis
;
Ultrasonography, Interventional
10.Time-Sequencing Morphometric Changes of Target Vessel Immediately after Percutaneous Coronary Balloon Angioplasty.
Dongsoo KIM ; Yangsoo JANG ; Hyuckmoon KWON ; Bum Kee HONG ; Hyun Seung KIM
Korean Circulation Journal 1998;28(2):222-229
BACKGROUND: Plaque compression (and/or redistribution) and vessel expansion are important mechanisms of percutaneous coroanry balloon angioplasty. We investigated the mechanisms of balloon angioplasty according to plaque characteristics by intravascular ultrasound and assessed the time-sequencing morphometric changes of target vessel after balloon dilation without catheter change using intravascular ultrasound balloon catheter. METHOD: We studied balloon angioplasty in 10 patients (eight male, average age of 55.3 years). Quantitative coronary angiography and intravascular ultrasound images were attained at baseline and at timed intervals of 0sec, 60sec and 180sec post-balloon angioplasty. The following categories were attained : reference diameter, minimal lumen diameter, cross sectional area (CSA) of lumen (L), external elastic membrane (EEM), and plaque + media (P+M). We also assessed the plaque morphology of target lesion and classified them into two groups according to intravascular ultrasound imaging : a soft plaque group versus a group characterized by fibrous and/or mildly calcified plaque. RESULTS: The proportions of plaque compression in the total luminal gain were 80% in the soft plaque group and 70% in the other ; the absolute amount of plaque compression was 26.9% in soft plaque and 24.0% in the other group. The time sequencing changes of target lesion EEM CSA of both group were 14.4+/-2.9mm2, 14.3+/-3.8mm2 (baseline) 15.1+/-2.5mm2, 15.4+/-3.7mm2 (immediate) 15.0+/-2.8mm2, 14.5+/-3.9mm2 (180sec), those of P+M CSA (target lesion) were 10.4+/-3.3mm2, 10.7+/-2.4mm2 (baseline) 7.6+/-2.7mm2, 8.1+/-2.4mm2 (immediate) 7.9+/-2.9mm2, 8.5+/-3.4mm2 (180sec). Target lesion lumen CSA were 4.0+/-1.1mm2, 3.6+/-2.0mm2 (baseline) 7.5+/-1.1mm2, 7.3+/-3.2mm2 (immediate) 7.1+/-1.3mm2, 6.0+/-1.7mm2 (180sec) respectively. CONCLUSION: Plaque compression (and/or redistribution) is the predominant mechanism of luminal gain in both groups. The absolute amounts of P+M CSA changes and time sequencing increment of target lesion were similar in both groups. In the non-soft group, the immediate increment and time sequencing reduction of EEM CSA in target lesion were greater than those of the soft plaque group.
Angioplasty
;
Angioplasty, Balloon
;
Angioplasty, Balloon, Coronary*
;
Catheters
;
Coronary Angiography
;
Humans
;
Male
;
Membranes
;
Phenobarbital
;
Ultrasonography