No abstract available.
Angioedema*

No abstract available.
Angioedema*

No abstract available.
Angioedema*

No abstract available.
Angioedema ; Eosinophilia

No abstract available.
Angioedema* ; Fluoxetine* ; Urticaria*

Studies on uticaris and quincke edema due to allergy of medications at the ward of Allergology and clinical immunology of Bach mai hospital (1991-2000). Result showed that: almost medications are used in medical clinic can cause allergic reactions. There were 393 kinds of medication that caused allergic reaction on 375 patients. In 23 different groups of medication. Antibiotics were main cause (67.2%) of allergic reactions, Beta-lactam class is 60.6% in the antibiotic group. besides urticaria and Quincke edema, there are many other clinical symptoms of allery from 375 in-patients: fever, erythema, dermatitis, dispnea. Allergic reactions were happened early (before 24 hours). Elevated ESR, white blood cell are main changes of teast. It takes short time to treat allergic patients (7.9 to 5.6 days). Solution of Methyl-Prednisolon, Dimedrol, Ascorbic acid and glucose 5% are common medications to treat this patients. No death occurred in patients of urticaria and Qincke edema during study periof.
Angioedema ; Hypersensitivity ; Edema

No abstract available.
Angioedema* ; Angioedemas, Hereditary* ; Humans ; Omalizumab*

Hereditary angioneurotic edema (HAE) is an autosomal dominant disease that results from the deficiency of C1 esterase inhibitor (C1-INH) function. Urgent proper intervention of the upper airway is warranted to save the life of patients with the disease, which often causes acute airway obstruction. We present, with a review of literature, a case of 37-year old woman with HAE of larynx.
Adult ; Airway Obstruction ; Angioedema ; Angioedemas, Hereditary* ; Female ; Hereditary Angioedema Types I and II ; Humans ; Larynx*

Recurrent cutaneous eosinophilic vasculitis (RCEV) is a rare cutaneous disease characterized by necrotizing vasculitis of small vessels, with exclusive eosinophilic infiltration and minimal, or no leukocytoclasis. It presents with recurrent erythematous or purpuric papules or plaques, or angioedema associated with peripheral eosinophilia. This disease follows a benign chronic course without evidence of systemic involvement. Systemic corticosteroid therapy is effective but the disease commonly pursues a relapsing and remitting course. Herein, we report a case of RCEV that was effectively treated with systemic corticosteroids and dapson(R)(diaminodiphenylsulphone). Furthermore, the patient did not show a relapse of the disease in spite the tapering the systemic corticosteroid dose.
Adrenal Cortex Hormones ; Angioedema ; Eosinophilia ; Eosinophils* ; Humans ; Recurrence ; Vasculitis*

Hypereosinophilic syndrome is a multisystem disorder with unexplained persistent eosinophilia and marked preference for systemic organ involvement. More than half of all patients have cutaneous involvement, usually presenting as angioedema, urticarial lesions, and eczematous, pruriginous papules. Hypereosinophilic syndrome presenting with generalized cutaneous blisters is rare, and we report the findings of a patient who was successfully treated with interferon-alpha.
Angioedema ; Blister* ; Eosinophilia ; Humans ; Hypereosinophilic Syndrome* ; Interferon-alpha