1.Transient Global Amnesia Caused by Bilateral Medial Temporal-Lobe Infarction.
UnKyu YUN ; Inha HWANG ; Sang Won HA
Dementia and Neurocognitive Disorders 2017;16(4):132-133
No abstract available.
Amnesia, Transient Global*
;
Infarction*
2.Transient Global Amnesia after Gastroscopy
Kyu On JUNG ; Hyeongsuk KIM ; Hye Jin MOON
Journal of the Korean Neurological Association 2018;36(3):241-242
No abstract available.
Amnesia, Transient Global
;
Gastroscopy
3.Repetitive Questioning and Writing in a Patient with Transient Global Amnesia.
Dae Lim KOO ; Jin Ah KIM ; Hyunwoo NAM
Journal of Clinical Neurology 2017;13(4):424-425
No abstract available.
Amnesia, Transient Global*
;
Humans
;
Writing*
4.Transient Global Amnesia Developed after Zolpidem Intake
Jean Hee KIM ; Yooha HWANG ; Byung Seok KIM ; Joong Seok KIM
Journal of the Korean Neurological Association 2018;36(3):226-230
We report two patients who complained of transient anterograde amnesia and repetitive questioning, that developed after taking zolpidem and lasted for several hours. The clinical manifestations of these patients fulfill the clinical criteria for transient global amnesia (TGA). The typical clinical manifestation of TGA following the consumption of zolpidem suggests a possible relationship associated with its drug mechanism.
Amnesia, Anterograde
;
Amnesia, Transient Global
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Humans
5.Seasonal Variation in the Incidence of Transient Global Amnesia in South Korea.
SangHak YI ; Young Ho PARK ; SangYun KIM
Journal of Clinical Neurology 2017;13(4):435-436
No abstract available.
Amnesia, Transient Global*
;
Incidence*
;
Korea*
;
Seasons*
6.A Case of Posterior Reversible Encephalopathy Syndrome with Similar Symptoms as Transient Global Amnesia.
Jeongyeon KIM ; Young Ik JUNG ; Junghee SEO ; Heejin LEE ; Mun Kyung SUNWOO
Dementia and Neurocognitive Disorders 2018;17(4):176-178
No abstract available.
Amnesia, Transient Global*
;
Posterior Leukoencephalopathy Syndrome*
7.Ischemic Evidence of Transient Global Amnesia: Location of the Lesion in the Hippocampus.
Youngsoon YANG ; Sangyun KIM ; Jae Hyoung KIM
Journal of Clinical Neurology 2008;4(2):59-66
Background and purpose: Transient global amnesia (TGA) is a rare amnestic syndrome characterized by the sudden onset of a selective anterograde and retrograde amnesia with a time course of up to 24 hours. Recent studies have found a high frequency of small high-signal abnormalities in the hippocampus on diffusion-weighted imaging (DWI), and accordingly ischemia has been proposed as an etiology of TGA. We hypothesized that TGA lesions occur preferentially in the CA1 region of the hippocampus, which is known to be susceptible to ischemia. Methods: Twenty consecutive patients with a clinical diagnosis of TGA underwent DWI both within 24 hours of symptom onset and 3 days later. Twenty patients with high-signal abnormalities in the hippocampus on the initial DWI underwent subsequent DWI and T2-weighted imaging in the coronal plane to precisely localize the lesions. Results: Seventeen patients had small high-signal abnormalities (with diameters of 1-3 mm) in the hippocampus unilaterally on DWI. One of these patients had two lesions in one hippocampus. Three of the 20 patients had lesions bilaterally in the hippocampus, 1 of whom had 3 bilateral lesions. A total of 25 lesions were identified: 5 in the hippocampal head, 19 in the body, and 1 in the tail. Six patients had unilateral lesions on the left,11 patients had them on the right, and 3 patients had bilateral lesions. Conclusions: In this study, lesions associated with TGA were localized mostly to the lateral portion of the hippocampus, corresponding to CA1. This finding supports the ischemic etiology of TGA, but the underlying pathophysiologic mechanism requires further investigation.
Amnesia, Retrograde
;
Amnesia, Transient Global
;
Head
;
Hippocampus
;
Humans
;
Ischemia
8.A Clinical Study of Transient Global Amnesia.
Journal of the Korean Neurological Association 1988;6(2):139-144
I studied 10 patients with transient global amnesia (TGA). On the C-T Brain Scan, 8 patients were normal and 1 patient had an old infarction in the right cerebral hemisphere and 1 patient had right medial thalamic hemorrhage. Electronencephalography was taken in 8 patients who were normal in C-T Brain Scan and the results were normal in all patients. Durations of amnesia were no longer than 6 hours in all patients except 1 patient that had amnesia for 6 days. Associated conditions and diseases with TGA patients were hypertension (in 5 patients), hypertrigliceridemia (in 4 Patients), cardiac arrthmia (in 2 patients), transient ischemic attack (in 1 patient) and diabetes mellitus (in 1 patient).
Amnesia
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Amnesia, Transient Global*
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Brain
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Cerebrum
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Diabetes Mellitus
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Hemorrhage
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Humans
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Hypertension
;
Infarction
;
Ischemic Attack, Transient
9.Seasonal Changes in the Incidence of Transient Global Amnesia.
Ophir KERET ; Nirit LEV ; Tzippy SHOCHAT ; Israel STEINER
Journal of Clinical Neurology 2016;12(4):403-406
BACKGROUND AND PURPOSE: Transient global amnesia (TGA) is a stereotypic condition characterized by anterograde and retrograde amnesia that typically resolves within 24 hours. The pathophysiology of TGA is still unclear. We noted that patients hospitalized with TGA tend to appear in seasonal clusters, and decided to investigate this phenomenon. METHODS: Every patient with acute presentation of amnesia at our medical center is hospitalized for observation and evaluation. We reviewed the monthly occurrence of TGA in our patient population between 2000 and 2014, and compared this to non-TGA hospitalizations during the same time period. RESULTS: During the analysis period, 154 patients who met the criteria for TGA were hospitalized, as well as 259,007 non-TGA hospitalizations. The annual occurrence of TGA ranged from 5 to 16 hospitalizations. There were 91 TGA events in women and 63 in men, in subjects aged 62.8±10.6 years (mean±SD). The incidence was maximal during December [odds ratio (OR)=2.83, 95% confidence interval (CI)=1.20–6.67] and March (OR=2.77, 95% CI=1.17–6.56), and minimal from April to August. The incidence exhibited an increase followed by a decrease from October to February. A seasonal trend was observed as well, with incidence peaks occurring in winter (OR=1.82, 95% CI=1.12–2.96) and spring (OR=1.80, 95% CI=1.10–2.94). CONCLUSIONS: Our findings suggest that the incidence of TGA exhibits seasonal variations. This observation may help to improve the understanding of the pathophysiology underlying TGA.
Amnesia
;
Amnesia, Retrograde
;
Amnesia, Transient Global*
;
Female
;
Hospitalization
;
Humans
;
Incidence*
;
Male
;
Seasons*
10.Recurrent Transient Global Amnesia in a Hot and Humid Machinery Room.
Kyung Hyun MIN ; Ji Seon KIM ; Sang Soo LEE ; Sung Hyun LEE ; Dong Ick SHIN ; Seo Young CHOI ; Minju YEO ; Inha KIM ; Sejin YANG ; Jin Hyun KIM
Journal of the Korean Neurological Association 2015;33(1):33-35
Transient global amnesia (TGA) is characterized by abrupt onset temporary dysfunction of anterograde and retrograde amnesia without other neurologic deficits. We encountered a 53-year-old man who developed recurrent TGA while working in a hot and humid machinery room (33degrees C and 64% relative humidity). Heat exposure and physical exertion may facilitate the leakage of cytokines into the systemic circulation so as to cause a cerebral endothelial insult. Functional insufficiency of the hippocampus and its connections caused by physical and environmental factors may be related to recurrent attacks.
Amnesia, Retrograde
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Amnesia, Transient Global*
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Cytokines
;
Hippocampus
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Hot Temperature
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Humans
;
Middle Aged
;
Neurologic Manifestations
;
Physical Exertion