1.Cerebrovascular ischaemia after carbon monoxide intoxication.
Hasan KARA ; A BAYIR ; Ahmet AK ; Selim DEGIRMENCI
Singapore medical journal 2015;56(2):e26-8
Carbon monoxide intoxication is the most prevalent cause of death from carbon monoxide poisoning. We herein report the case of a 56-year-old man who was found unconscious and smelled of smoke after exposure to carbon monoxide from a heater. He scored 5 on the Glasgow Coma Scale, and had respiratory insufficiency and elevated troponin I, creatine kinase-MB fraction and carboxyhaemoglobin levels. He was treated by mechanical ventilation. After regaining consciousness, brain magnetic resonance imaging showed diffusion restriction in the left occipital lobe; there was a loss of vision (right temporal hemianopsia), which improved by the follow-up session. Carbon monoxide intoxication may cause neurologic and cardiac sequelae, and the initial treatment includes oxygen therapy. Acute carbon monoxide poisoning can cause serious injury to the brain, heart and other organs; the most severe damages that could be inflicted to the brain include cerebral ischaemia and hypoxia, oedema, and neural cell degeneration and necrosis.
Brain
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physiopathology
;
Brain Ischemia
;
physiopathology
;
Carbon Monoxide
;
chemistry
;
Carbon Monoxide Poisoning
;
physiopathology
;
Carboxyhemoglobin
;
chemistry
;
Creatine Kinase, MB Form
;
blood
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Diffusion
;
Glasgow Coma Scale
;
Humans
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Hyperbaric Oxygenation
;
Hypoxia
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Magnetic Resonance Imaging
;
Male
;
Middle Aged
;
Myocardial Ischemia
;
physiopathology
;
Stroke
;
physiopathology
;
Troponin I
;
blood
2.Histopathologic Changes in Dental Follicle Associated with Radiographically Normal Impacted Lower Third Molars
Ahmet Altan ; Sefa Ç ; olak ; Elif Akç ; ay ; Nihat Akbulut
Archives of Orofacial Sciences 2023;18(no.1):19-26
Impacted third molar extractions are one of the most common oral surgical procedures. In cases
where a pathological condition is observed, it is relatively easy to decide on surgery. However, cystic
changes can be observed in follicular tissues even in cases where pathological changes are not observed on radiography. The present study aimed to evaluate the histopathologic changes in dental follicles associated with radiographically normal impacted lower third molar. One hundred and one dental follicles were obtained after third molar surgeries. The patients with follicular width of less than 2.5 mm in the panoramic radiographs were included in the study. The relationships between pathological changes in follicular tissues and angular position of impacted teeth, age and gender were statistically examined. Cystic changes were observed in 65 (64.3%) of 101 follicles examined. The relationship between cystic changes and the angular position of impacted lower third molars was statistically significant (p < 0.05). Cystic changes were observed in 13 (43.3%) of 30 mesioangular teeth, 24 (80%) of 30 vertical teeth, 19 (65.5%) of 29 horizontal teeth, and 9 (75%) of 12 distoangular teeth. About 60% of cystic changes and 64.06% of inflammation occurred in patients between the ages of 20 and 30. Cystic changes can be observed in the follicular tissues of impacted lower third molars that are radiographically normal. There is a probability of histopathological changes, especially in patients over the age of 20 years and in the impacted lower third molars in the vertical position.