1.A case of Distal Renal Tubular Acidosis.
Seong Gyoo PARK ; Gyung Min ANN ; Sang Hee PARK ; Kwang Chul LEE ; Chang Sung SON ; Pyung Hwa CHOI
Journal of the Korean Pediatric Society 1990;33(4):539-543
No abstract available.
Acidosis, Renal Tubular*
2.A case of type 4 renal tubular acidosis resulting from captopril administration.
Jeong Eun PARK ; Gyu Bog CHOI ; Kyun Il YOON
Korean Journal of Nephrology 1991;10(4):620-624
No abstract available.
Acidosis, Renal Tubular*
;
Captopril*
3.Two cases of distal renal tubular acidosis associated with immune-mediated diseases.
Korean Journal of Medicine 1993;45(5):664-669
No abstract available.
Acidosis, Renal Tubular*
4.Distal renal tubular acidosis in sjogren syndrome with rheumatoid arthritis.
Jun Sang LEE ; Sung Il KIM ; Yong Seok YANG ; Moo Young KIM ; Il Doo LEE ; Young Soo KIM ; Ihm Su KWAK ; Ha Youn RHA
Korean Journal of Nephrology 1993;12(4):732-736
No abstract available.
Acidosis, Renal Tubular*
;
Arthritis, Rheumatoid*
;
Sjogren's Syndrome*
5.A case of type 4 renal tubular acidosis associated with systemic lupus erythematosus.
Hae Ok JUNG ; Jee Won PARK ; Seung Joon KIM ; Dong Chan JIN ; Young Soo KIM ; Young Suk YOON ; Byung Kee BANG ; Young Jin CHOI
Korean Journal of Nephrology 1993;12(4):724-731
No abstract available.
Acidosis, Renal Tubular*
;
Lupus Erythematosus, Systemic*
6.A case of distal type of renal tubular acidosis in a neonate.
Sung Sub SHIM ; Young Joon KIM ; Jae Hong PARK ; Soo Yung KIM ; Chan Yung KIM
Journal of the Korean Pediatric Society 1992;35(7):1014-1018
No abstract available.
Acidosis, Renal Tubular*
;
Humans
;
Infant, Newborn*
7.A Case of Distal Renal Tubular Acidosis Associated with Medullary Sponge Kidney.
Sung Jun JANG ; Jo Yun JUNG ; Jun Sik KIM ; Heung Sik KIM ; Hee Jung LEE
Journal of the Korean Pediatric Society 2001;44(1):89-93
Renal tubular acidosis is a clinical state of systemic hyperchloremic acidosis resulting from impaired urine acidification. Medullary sponge kidney is a renal parenchymal malformation characterized by cystic dilatation of the collecting ducts. Although medullary sponge kidney is a congenital disease, it is rarely identified in childhood and is usually discovered in adulthood. Medullary sponge kidney patients may have defects in urinary acidification and concentration mechanism. We experienced a case of distal renal tubular acidosis associated with medullary sponge kidney. So, we report a case of distal renal tubular acidosis associated with medullary sponge kidney with a brief review of the related literature.
Acidosis
;
Acidosis, Renal Tubular*
;
Dilatation
;
Humans
;
Medullary Sponge Kidney*
8.Acetazolamide-Induced Type II Renal Tubular Acidosis and Muscle Weakness
Journal of the Korean Neurological Association 2019;37(4):420-422
No abstract available.
Acetazolamide
;
Acidosis
;
Acidosis, Renal Tubular
;
Hypokalemic Periodic Paralysis
;
Muscle Weakness
9.Renal tubular acidosis in Papua New Guinea
Papua New Guinea medical journal 1994;37(1):45-49
Unlike most other inborn errors of metabolism, which require advanced and expensive diagnostic techniques and complex drug and dietary management (often not feasible in developing countries), the renal tubular acidoses may be detected and treated both easily and cheaply. Diagnostic confusion is possible as this series demonstrates due to the protean clinical manifestations. Three recent cases from Port Moresby General Hospital are described and appropriate investigations and treatment discussed.
Acidosis, Renal Tubular - diagnosis
;
Acidosis, Renal Tubular - metabolism
;
Child, Preschool
;
Female
;
Humans, Infant
10.Metabolic acidosis and urinary acidification defect during the course of hemorrhagic fever with renal syndrome
Jin Suk HAN ; Gheun Ho KIM ; Jaeho EARM ; Kwon Wook JOO ; Wooseong HUH ; Un Sil JEON ; Curie AHN ; Suhnggwon KIM ; Jung Sang LEE
Journal of Korean Medical Science 1998;13(4):389-394
To evaluate urinary acidification defect and its contribution to metabolic acidosis (MA) during hemorrhagic fever with renal syndrome (HFRS), we serially analyzed acid-base balance and urinary acidification indices in 10 HFRS patients. Data of the patients were compared with those of 8 normal volunteers (NC). MA was observed in 6 of 8 patients in the oliguric phase, 5 of 7 in the early diuretic phase, 8 of 10 in the late diuretic phase and 2 of 9 in the convalescent phase. HFRS patients with MA had a higher plasma anion gap in the oliguric and early diuretic phases than NC and a higher plasma Cl/Na ratio in the late diuretic phase than NC. As compared with acid-loaded NC, HFRS patients had a higher urine pH in the oliguric, early diuretic and late diuretic phases, a higher urine anion gap (UAG) in the oliguric and early diuretic phases and a lower urinary NH4+ excretory rate in the oliguric, early diuretic and late diuretic phases. Overt distal acidification defect was observed in 6 of 8 patients in the oliguric phase, 3 of 7 in the early diuretic phase, 5 of 10 in the late diuretic phase and none of 9 in the convalescent phase. None of the convalescent patients had latent acidification defect. In conclusion, urinary acidification defect is marked in the oliguric and diuretic phases of severe HFRS and may play a role in the development of a high anion gap (AG) metabolic acidosis in the earlier phase and hyperchloremic MA in the later phase, but rapidly recovers in the convalescent phase.
Acidosis, Renal Tubular/urine
;
Acidosis, Renal Tubular/metabolism
;
Hemorrhagic Fever with Renal Syndrome/urine
;
Hemorrhagic Fever with Renal Syndrome/metabolism
;
Human