No abstract available.
Balloon Valvuloplasty* ; Pulmonary Valve Stenosis* ; Pulmonary Valve*

No abstract available.
Pulmonary Valve* ; Tetralogy of Fallot*

No abstract available.
Pulmonary Valve* ; Tetralogy of Fallot*

No abstract available.
Mitral Valve* ; Pulmonary Edema* ; Rupture*

No abstract available.
Mitral Valve Stenosis* ; Pulmonary Infarction*

Repair of tetralogy of Fallot (TOF) has shown excellent outcomes. However it leaves varying degrees of residual hemodynamic impairment, with severe pulmonary stenosis (PS) and free pulmonary regurgitation (PR) at both ends of the spectrum. Since the 1980s, studies evaluating late outcomes after TOF repair revealed the adverse impacts of residual chronic PR on RV volume and function; thus, a turnaround of operational strategies has occurred from aggressive RV outflow tract (RVOT) reconstruction for complete relief of RVOT obstruction to conservative RVOT reconstruction for limiting PR. This transformation has raised the question of how much residual PS after conservative RVOT reconstruction is acceptable. Besides, as pulmonary valve replacement (PVR) increases in patients with RV deterioration from residual PR, there is concern regarding when it should be performed. Regarding residual PS, several studies revealed that PS in addition to PR was associated with less PR and a small RV volume. This suggests that PS combined with PR makes RV diastolic property to protect against dilatation through RV hypertrophy and supports conservative RVOT enlargement despite residual PS. Also, several studies have revealed the pre-PVR threshold of RV parameters for the normalization of RV volume and function after PVR, and based on these results, the indications for PVR have been revised. Although there is no established strategy, better understanding of RV mechanics, development of new surgical and interventional techniques, and evidence for the effect of PVR on RV reverse remodeling and its late outcome will aid us to optimize the management of TOF.
Dilatation ; Heart Failure ; Hemodynamics ; Humans ; Hypertrophy ; Mechanics ; Pulmonary Valve ; Pulmonary Valve Insufficiency ; Pulmonary Valve Stenosis ; Tetralogy of Fallot

No abstract available.
Heart Septal Defects, Ventricular* ; Pulmonary Valve Stenosis*

The remote location of VSD relative to the aortic valve imposes considerable surgical difficulties in the repair of DORV with noncommitted VSD. We report a successful biventricular repair of the anomaly with VSD rerouting to pulmonary artery followed by arterial switching operation.
Aortic Valve ; Heart Defects, Congenital ; Pulmonary Artery

From March 1995 to May 2002, at Hanoi Military Hospital N.108,on 98 patients taken surgical commissurotomy for mitral valve, pulmonary arterial pressure declined from 40,7± 12,7 mmHg before surgery to 29,6 ± 8,2mmHg after surgery, right ventricular pressure from 38,1 ± 11,1mmHg to 29,4 ± 8,4mmHg and left auricular pressure from 32,2 ± 8,6mmHg to 23,2± 7,1mmHg. This had demonstrated very good results of mitral valve commissurotomy, in l to improve hemodynamic state in pulmonary arterial pressure and heart chamber pressure
Pulmonary Wedge Pressure ; heart ; Mitral Valve ; surgery ;

Tetralogy of Fallot (TOF) is an index lesion for all paediatric and congenital heart surgeons. In designing an appropriate operation for children with TOF, the predicted postoperative physiology must be taken into account, both for the short and long term. A favourable balance between pulmonary stenosis (PS) and pulmonary insufficiency (PI) may be critical for preservation of biventricular function. A unified repair strategy to limit both residual PS and PI is presented, along with supportive experimental evidence. A strategy for dealing with coronary anomalies and some comments regarding best timing of operation are also included.
Child ; Heart ; Heart Ventricles ; Humans ; Pulmonary Valve ; Pulmonary Valve Stenosis ; Tetralogy of Fallot