No abstract available.
Headache ; Meningeal Carcinomatosis* ; Papilledema*

No abstract available.
Child* ; Humans ; Papilledema*

A rare case of a giant-celled glioblastoma arised from the intracranial portion of the right frontal base is presented. Clinical presentation was that of increased intracranial pressure with bilateral optic papilledema. Skull X-rays showed an erosion of the right orbital roof. Right carotid angiogram confirmed the presence of a tumor in the frontal base.
Cerebrum* ; Glioblastoma* ; Intracranial Pressure ; Orbit ; Papilledema ; Skull

No abstract available.
Behcet Syndrome ; Optic Nerve Diseases ; Papilledema

No abstract available.
Child* ; Humans ; Papilledema ; Pseudotumor Cerebri* ; Stents*

Objective: We reported the association between iron-deficiency anemia and papilledema in a case of presumed idiopathic intracranial hypertension (IIH) and subsequent resolution of the signs and symptoms on correction of anemia. Methods: Case notes, blood tests, clinical photographs, and neuroimaging were collected and analyzed. Iron-deficiency anemia was treated with oral ferrous sulphate. Results: Blood tests revealed a microcytic anemia consistent with iron-deficiency anemia. Magnetic resonance imaging did not demonstrate enlarged ventricles. There was complete resolution of all signs and symptoms after treatment of the anemia. Conclusion We recommend that a simple full blood count should be performed on all patients diagnosed with IIH.
Anemia, Iron-Deficiency ; Papilledema ; Pseudotumor Cerebri

A case is reported which raised intracranial pressure with papilledema due to hydrocephalus associated with a intramedullary spinal cord tumor at the T10-T12 levels. The pathlogical diagnosis of the tumor is pleomorphic xanthoastrocytoma which is very rare in the spinal cord. CSF protein was elevated. The mechanisms of the raised intracranial pressure are discussed with a review of the literatures.
Diagnosis ; Hydrocephalus* ; Intracranial Pressure ; Papilledema ; Spinal Cord Neoplasms* ; Spinal Cord*

The clinical syndrome of acute optic neuritis(ON) has been known for many yearsthough its causes are still uncertain. Especially, the main focus is how often after a firstbout of isolated ON progress to multiple sclerosis in neurologic field. Therefore westudied and analysed 20 patients with acute optic neuritis for clinical features and prognosis and obtained the results as follows; 1. The male to female ratio was 1: 4. 2. The age at onset ranged from 9 to 70 years. 3. Unilateral involvement -of ON was detected in 10 cases (50%); the others were bilater al(10 cases, 50%). 4. Papillitis was present in 6 cases(30.2%) and the disk was normal(retrobulbar) in the other 7 cases (35%). 5. Recovery of vision was noted within 2 weeks in 8 eyes(34.8%) and in 19 eyes (82.2%) within 2 months. 6. The response to treatment in optic neuritis showed no definite difference between steroid group and conservative group. 7. The rate of ON evolving into multiple sclerosis was 10% in this series.
Female ; Humans ; Male ; Multiple Sclerosis ; Optic Neuritis* ; Papilledema ; Prognosis*

Papilledema, pupillary abnormalities, and nystagmus are common neuro-ophthalmologic signs in neurocysticercosis (NCC). Oculomotor palsy rarely occurs and usually accompanies compression of the midbrain by supratentorial or subarachonoid lesions with or without inflammation and hydrocephalus. Oculomotor palsy from NCC involving the midbrain parenchyme has rarely been described. We report on a patient who presented with oculomotor palsy caused by mesencephalic NCC. The patient showed recurrences of symptoms in association with steroid tapering.
Humans ; Hydrocephalus ; Inflammation ; Mesencephalon* ; Neurocysticercosis* ; Papilledema ; Paralysis* ; Recurrence

PURPOSE: To investigate the relationship between chorioretinal lesions and papilledema in patients with hypertensive optic neuropathy. METHODS: We analyzed the angiographic findings of 142 patients (284 eyes) who had been diagnosed with hypertensive chorioretinal disease in the ophthalmic outpatient department of our hospital, from March 2001 to December 2003. We classified hypertensive ocular disorders to either the optic disc edema group or the non-optic disc edema group. RESULTS: There were 34 patients with hypertensive optic neuropathy. With an analysis of color photo and angiographic findings, a significant correlation of optic disc edema was seen in Elschnig's spot, prominent watershed zone, and retinal hemorrhage. CONCLUSIONS: Hypertensive optic neuropathy was significantly related to retinal hemorrhage, Elschnig's spot, and prominent watershed zone. We should carefully observe the patients who have these lesions.
Edema ; Humans ; Hypertension ; Optic Nerve Diseases* ; Outpatients ; Papilledema ; Retinal Hemorrhage