Background: Left Ventricular Hypertrophy (LVH) has been shown to be a risk factor as well as a consequence of cardiovascular diseases. The importance of an early LVH diagnosis in the community has been desmontrated by many case studies. Objectives: (1) Find out the prevalence of electrocardiographic LVH. (2) Determine the risk factors of electrocardiographic LVH. Subjects and method: 3561 people (1410 men) over 25 years old were chosen from 3 provinces/cities (Ha Noi, Thai Binh, and Nghe An). All participants underwent electrocardiogram, and the Romhilt-Estes score on standard 12-lead electrocardiogram to measure LVH. Diagnosis of LVH was made when Romhilt-Estes score >=4. Data was analyzed by Epi-Info software version 6.04 and SPSS version 13.0. Results: According to JNC 7, there were 744 people with hypertension (352 men), accounted for 21.2%. The rate of hypertension was 24.9% in men and 18.2% in women. This difference was statistically significant (p <0.001). Overall prevalence of electrocardiographic LVH was 6.71% (95% CI: 5.94 \u2013 7.58%). Prevalence was 9.00% among men (95% CI: 7.62 \u2013 10.61%) and 5.21% in women (95% CI: 4.35 \u2013 6.23%). Risk factors of electrocardiographic LVH included male, elderly, hypertension, and obesity. Conclusion: The high prevalence of LVH in the population showed the importance of detection and early treatment for LVH patients, especially in those, who had no clinical symptoms.
Left ventricular hypertrophy ; electrocardiogram

A cross-sectional discriptive, compare and control study on the 125 patients with the old cardiac infarction and 55 healthy people was carriout in the Army Central Hospital No108 from 3/1995 to 3/1998. The age and sex between 2 groups were the same. The results have shown that the EF% calculated as improved simpson and the interval from point E of the anterior leaf of mitral valve to the left side of the ventricular connection Septal eirs were 2 parameters with the sensitivity and specificity higher than other parameters of ultrasound. The patients with the old cardiac infarction had significantly reduced the left ventricular systolic function. This reduction was corelated with the cardiac infarction. The most reduced the left ventricular systolic function found in the group of the cardiac infarction in the both anterior and posterior septal.
Ventricular Function, Left ; Infarction

The number of people identified as having Hypertension continues to increase and most of them have left ventricular hypertrophy- an earliest complication and also being a risk factor of heart disease. In clinical practice in Cardiovascular Department- Hue Central Hospital, until now, diagnosis LVH depends on ECG so it misses many cases as having a real LVH. This is the first time, we use Echocardiography to diagnose LVH in patients with Hypertension by using LVMI. In 56 cases of hypertension we found a larger number of LVH by using LVMI than by ECG (41cases Vs 22cases- p<0,01). This is an useful method in diagnosis for LVH.
Hypertension ; Hypertrophy, Left Ventricular

A study on 97 male patients with the hypertension ages of 64,1 +/- 14 in hospital 171 during 1993-5/1997 has shown that the hypertension was most frequently occurred in age of 60 (81,24%). The cardiac complications and large arterial complications in hypertension were common (67,01%) the cardiac complications include the left ventricular thickness (45,36% 45 cases with the left ventricular thickness were diagnosed by laparoscopy in which 30 cases with the centric thickness, 12 cases with the incentric thickness. The sensitivity and specificity of the eletrocardiography was 21/45 and 48/52, respectively. The frequent of the left ventricular thickness increased as increased age and its progress depends on the hypertension. The relation of the disorder of lipid metabolism and artherosclerosis exacerbated the hypertension and the left-ventricular thickness. The laparoscopy is valuable method diagnosis of the left ventricular thickness.
Hypertension ; Hypertrophy, Left Ventricular

Back ground: The hypertension may lead the changes of the left ventricular geometry and the left ventricular diastolic dysfunction. There were many investigations about these abnormalities. Moreover, the treatment of the hypertensive patients with or without the left ventricular diastolic dysfunction is quite different. In Binh dinh general hospital, however, the left ventricular dysfuntion were neglected in practice. Thus, in this study, we evaluate the prevalence of these abnormalities in hypertensive patients. Subjects and method: We prospectively studied, from 6/2002 to 11/2003, 200 consecutive hypertensive patients admitted to the Internal Deparment at the Binh Dinh General Hospital. Echocardiography was performed on a Hewlett-Packard Image-Points with 2.5 and 3.5 MHz transducers. Measurements of the size of the ventricular walls are according to ASE. Doppler echocardiography is used to evaluate the characteristics of diastolic transmitral blood flow from the apical four-chaber view. The peak velocities of blood flow during early diastolic filling (E wave) and atrial contraction (A wave), the E-wave decelerating time and parameters of pulmonary venous flow revesal during atrial contraction are measured. The isovolumic relaxation time is measured from the apical-five chamber view. The diagnosis and classification of the left ventricular geometric changes and the abnormal diastolic filling patterns are based on the criteria of Devereux. Results: Prevalence of the left ventricular diastolic dysfunction was 61.5% of the hypertensive patients with the most frequent impaired relaxation pattern (48%). 72.5% of patients have the geometric changes in which the remodelling type consists of 47.5% concentric hypertrophy 25%.
Ventricular Dysfunction, Left ; Hypertension

Study on 66 men with type 2 diabetic at Military Hospital 108 and 103 healthy persons (with similar age, BIM, HATTh, HATTr, frequence of heart). Results: In type 2 diabetic group, A-C distance front of bicuspid valve extend, slash volume significant reducing compared with control group. Left autrial size, intraventricular septum wall thickness, ventricular wall thickness, left ventricular mass index (LVMI) significant changing compared with control group. Non THA diabetic group include 3 patients ventricular hypertrophy (LVMI > 131 g/m2), in control group hasn’t any patients. In VT Ia diabetic group, VTIe/VTIa rate reduce, DT time lengthen significantly compare with control group
Diabetes Mellitus, Type 2 ; Ventricular Dysfunction, Left ; Ventricular Function, Left

No abstract available.
Atrial Function, Left ; Sprains and Strains

BACKGROUND AND OBJECTS: Mitral annulus velocity by Pulsed Wave Doppler has been used as method of evaluation of left ventricular diastolic function. However, it is unknown how this is altered in the patients with left ventricular hypertrophy (LVH). this study was aimed to compare a group of healthy subjects with a group of patients with LVH. METHODS: Subjects were 80 patients with LVH (left ventricular mass index125 g/m2) and 163 controls with normal left ventricular mass index (<125 g/m2). For measuring the mitral annulus velocities by doppler tissue imaging (=DTI), we used the 2.5 MHz probe (Sequoia, Accuson) in apical 4 chamber view with the sample volume at the septal portion of the mitral annulus. RESULTS: Mitral annular velocity in diastole is easily recorded by DTI in all subjects. in LVH group, mitral annulus E've#locity was significantly lower than controls (5.2+/-1.3 cm/sec vs 6.2+/-1.7 cm/sec, p(<0.001) and mitral annulus A' velocity was also significantly higher than controls (8.5+/-1.3 cm/sec vs 8.0+/-1.1 cm/sec, p<0.02). annular displacement measured by DTI-TVI (time velocity integral), also In LVH group, mitral annulus E'-TVI was significantly lower than controls (5.7+/-1.8 mm vs 6.8+/-2.2 mm, p<0.001). CONCLUSION: Mitral annulus velocity determined by DTI could be used as one of the parameters in evaluating diastolic function in patient with LVH.
Diastole ; Humans ; Hypertrophy, Left Ventricular*

No abstract available.
Humans ; Thrombosis* ; Ventricular Dysfunction, Left*

No abstract available.
Breast* ; Humans ; Ventricular Dysfunction, Left*