1.Haemodynamic changes in high altitude pulmonary edema and effects of oxygen breathing
Xinbing MU ; Suzhi LI ; Yuqi GAO ; Fuyu LIU ; Ganglin YE ; Hongya TANG ; Dan ZHU ; Xiaobo ZHOU ; Guangli CHEN
Chinese Journal of Pathophysiology 2000;0(08):-
AIM: To explore the pathogenic mechanism of high altitude pulmonary edema(HAPE). METHODS: Haemodynamic changes and effects of 100 percent oxygen breathing were measured by Swan-Ganz thermistor catheters, high altitude healthy volunteers were served as controls. RESULTS: The important features of haemodynamic changes in HAPE: (1)Pulmonary arterial pressure was raised; (2)Pulmonary arterial resistance and cardiac output were raised; (3)Pulmonary artery wedge pressures and right atrial pressure were normal; (4)Pulmonary arterial pressure and resistance were induced by oxygen breathing. CONCLUSIONS: The normal pulmonary artery wedge pressures with a high cardiac output indicated that HAPE was recognized as a form of noncardiogenic pulmonary edema. The pulmonary hypertension may play an important role in the development of HAPE.
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