1.Endovascular laser He-Ne and micro frequency electromagnetic stimulation in treatment of Parkinson in the Center for Biomedical Physic
Journal of Practical Medicine 2002;435(11):37-40
Parkinson is popular disease caused by degeneration of central nervous system. 17 patients received the specific drug therapy but had no effect. They then received the endovascular laser He - We in combination with micro frequency electromagnetic stimulation. The results have shown that the disease’ s symptoms were remised and dosage of current drugs was reduced. The method found simply, easy to implement and no effects on the cardiovascular system
Parkinson Disease
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Electromagnetics
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lasers
2.Study of the Pulse Waveform and the Design of the Portable, Non-invasive, Electromagnetic Stimulation for Enhancement of Fracture Healing.
Young Hoo KIM ; Won Ki KIM ; Sung Jae KIM
Yonsei Medical Journal 1984;25(2):149-157
Since the concept of bioelectrical potentials in the bone was developed, various electrical systems have been utilized to heal non-united fractures. The asymmetrical and time varied pulsed electromagnetic stimulator was designed and utilized to treat eight fractures of the lower extremity and one congenital pseudoarthrosis of the tibia. Nonunited fractures, neglected old fractures, and a case of congenital pseudoarthrosis responded favorably to our designed pulse waveform. On the other hand, fresh fractures responded equivocally. A new1y designed electromagnetic stimulator which can deliver vascular and calcification pulses, will be developed on an industrial basis to provide various clinical and experimental applications.
Electromagnetics/instrumentation
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Electromagnetics/therapeutic use*
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Equipment Design
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Fractures/therapy*
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Human
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Leg Injuries/therapy*
3.Magnetic stimulation accelerating rehabilitation of peripheral nerve injury.
Ahmed, BANNAGA ; Tiecheng, GUO ; Xingbiao, OUYANG ; Daosong, HU ; Chuanyou, LIN ; Fuyuan, CAO ; Yunping, DENG ; Zhengcheng, GUO ; Yongxiang, LUO
Journal of Huazhong University of Science and Technology (Medical Sciences) 2002;22(2):135-9
The effect of magnetic stimulation (MS) on sciatic nerve injury was observed. After sciatic nerve was crushed in 40 Sprague Dawley (SD) rats, one randomly selected group (group D) was subjected, from the 4th day post-operatively to 3 min of continuous 70% of maximum output of MS daily for 8 weeks. The other group (group E) served as a control group. The nerve regeneration and motor function recovery were evaluated by walking track analysis (sciatic function index, SFI; toe spreading reflex, TSR), electrophysiological, histological and acetylcholineesterase histochemistry. The SFI in the group D was greater than in the group E with the difference being statistically significant (P < 0.01). TSR reached its peak on the 4th day in the group D and on the 10th day in the group E respectively. The amplitude and velocity of MCAP and NCAP in the group D was greater than in the group E with the difference being statistically significant (P < 0.01), while the latency and duration of MCAP and NCAP in the group D were less than in the group E with the difference being also statistically significant (P < 0.01). Histological examination showed the mean axon count above the lesion for thick myelinated fibers (> 6.5 microns) in the group D was greater than in the control group with the difference being statistically significant (P < 0.01), while the mean axon count below the lesion for thick myelinated fibers was less than that in the group E with the difference being statistically significant (P < 0.01). The mean axon count above the lesion for thin myelinated fibers (2-6.5 microns) in the group D was greater than that in the group E with the difference being statistically significant (P < 0.05), while the mean axon count below the lesion for thin myelinated in the group D was greater than that in the group E with the difference being statistically significant (P < 0.01). Acetylcholine esterase examination showed that the MS could significantly increase the number of the motor neurons. There was no significant difference in the number of the motor neurons between the treatment side and the normal side (P > 0.05). It can be concluded that MS can enhance functional recovery and has a considerable effect in the treatment of the peripheral nerve injury.
Acetylcholinesterase/metabolism
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Electromagnetics
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Motor Neurons/physiology
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*Nerve Regeneration
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Random Allocation
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Rats, Sprague-Dawley
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Sciatic Nerve/*injuries
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Sciatic Nerve/*physiopathology
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Sciatic Neuropathy/rehabilitation