1.Fibrinolytic Response to Standardized Venous Occlusion in Cerebral Infarction Patients(A preliminary study).
Hyun Gil SHIN ; Dong Ho YANG ; Sae Yong HONG ; Kwang Ho LEE
Journal of the Korean Neurological Association 1992;10(4):509-514
No abstract available.
Cerebral Infarction*
2.Cerebral Infarction Caused by Painless Aortic Dissection
Dong Gyu LIM ; Jeong Hoon KIM ; Hye Yoon KIM ; Sang Soon PARK ; Jae Hyeok HEO
Journal of the Korean Neurological Association 2018;36(4):405-407
No abstract available.
Cerebral Infarction
4.Nasopharyngeal carcinoma presenting as a dual territory stroke: The hyperdense artery sign.
Philippine Journal of Otolaryngology Head and Neck Surgery 2011;26(2):37-38
This 63 year-old chinese female, with both diabetes and hypertension, underwent CT imaging of the brain after presenting with a progressive left sided hemiplegia.
The ‘hyperdense artery sign’ is a generic description that can be evident in any artery of the body on unenhanced CT, occurring due to the presence of intraluminal thrombosis (Figure 1). It is a well-established sign, most commonly described in CT imaging of the brain, where it is visualised in the vast majority of cases in the middle cerebral artery in the context of an acute cerebral infarction.1 It occurs uncommonly elsewhere, with the internal carotid artery (ICA) and basilar artery being other clinically significant sites. The ‘hyperdense ICA’ sign has been reported to be a reliable and highly specific marker of thromboembolic occlusion of the internal carotid artery.2 The ‘hyperdense artery sign’ is related to the attenuation value of intraluminal thrombus. The CT attenuation value (Hounsfield unit or HU) of normal blood is dependent on the haematocrit, ranging from 20 to 30 HU. As the process of thrombus retraction occurs, its water content decreases, increasing the concentration of haemoglobin within the clot. As a result this raises the attenuation value of the thrombus to 50–80 H. So the term ‘hyperdense’ is given.3
In this case, it proved to be the presenting symptom for an undiagnosed nasopharyngeal tumour, the thrombus likely developing as a complication of the surrounding tumour within the nasopharyngeal recess. The resultant outcome was a dual territory cerebral infarction of the anterior and middle cerebral artery territories, both supplied by branches of the internal carotid artery (Figures 2a & 2b).
Cerebral Infarction
5.A Case of Cerebral Cortical Infarction Presenting as Peripheral Pattern Wrist Drop.
In Uk SONG ; Min Sung KIM ; Du Shin JEONG ; Tae Kyeong LEE ; Gi Bum SUNG ; Moo Young AHN
Journal of the Korean Neurological Association 2002;20(4):439-441
No abstract available.
Cerebral Infarction
;
Infarction*
;
Wrist*
6.Clinical features and risk factors of cerebral lacunar infarction
Journal of Practical Medicine 2002;437(12):35-36
A study on 60 patients with the cerebral lacunar infarction, ages of 20-85 and 60 patients with cerebral infarction (control), ages of 20 -87 has shown that the syndromes of cerebral lacunar infarction comprised hemiplegia type of merely moving, speaking disorder hemiplegia type of combination of sense and moving, merely sense accident and other lacunar syndromes. The risk factors of diseases were hypertension, diabetes and smoking.
Cerebral Infarction
;
Brain Infarction
;
diagnosis
7.The relationship between hyperhomocysteinemia and cerebral infarction due to large and small artery thrombosis
Ho Chi Minh city Medical Association 2005;10(3):147-150
A study was conducted on 220 patients with the first ischemic infarction due to atherosclerosis treated at Stroke Unit of People Hospital No 115 from January 2002 to April 2002, and 230 control subjects. Results: Among 220 cases of ischemic infraction, there were 112 female and 108 male, mean ages 60.71 ±11.9 years old. Among them, 20% is due to large artery thrombosis and the mean plasma homocystein levels was 14.4µmol/L, 80% is due to small artery thrombosis and the mean plasma homocystein levels is 13µmol/L. There was no significantly difference in the mean plasma homocystein levels between large artery thrombosis and small artery thrombosis (p=0.1). The adjusted odd ratios (OR) for large artery and small artery infarctions associated with moderate hyperhomocysteinemia (>1515µmol/L) were 2.56 and 2.20, respectively
Cerebral Infarction
;
Arteries
;
Hyperhomocysteinemia
8.Research on the effect of electro boa acupuncture and electro acupuncture to rehabilitate motor function for hemiplegic patient caused by cerebral infarction
Journal of Medical Research 2005;39(6):51-55
Electro boa acupuncture (EBA) and electro acupuncture (EA) to rehabilitate the motor function for hemiplegic patients caused by cerebral infarction. Objective: (1) Describe the clinical characteristic of hemiplegic patients caused by cerebral infarction. (2) Evaluate the rehabilitation of motor function in Hemiplegic patients acording to clinical characteristics. Method: 100 hemiplegic patients was selected accidentaly. 50 hemiplegic patients caused by cerebral infarction was applied EBA , and 50 hemiplegic patients was applied EA. The result of treatment was compared after 30 days Resuls: Majority of patien was over 60 years old. In EBA group very good result (recovery) was 30%, good effect 56%; in EA group very good result (recovery) was 18%, good effect 58%. According to Orgogozo and Barthel scale, after treatment, group of patients with EBA, the indexes are all higher than group with EA. Conclusion: EBA to rehabilitate the motor function for hemiplegic patients caused by cerebral infarction has better results than EA.
Cerebral Infarction, Electroacupuncture, Therapeutics
9.Cerebral Metastatic Myxoma in a Cerebral Infarction Patient after Complete Resection of Cardiac Myxoma.
Jae Chan RYU ; Jee Hyun KWON ; Da Young LEE ; Dong Suk YANG ; Wook Joo KIM ; Misung KIM
Journal of the Korean Neurological Association 2017;35(4):254-256
No abstract available.
Cerebral Infarction*
;
Humans
;
Myxoma*
10.Multifocal Myoclonus as a Manifestation of Acute Cerebral Infarction Recovered by Carotid Arterial Stenting.
Hyangkyoung KIM ; Jun Soo BYUN ; Mark HALLETT ; Hae Won SHIN
Journal of Movement Disorders 2017;10(1):64-66
No abstract available.
Cerebral Infarction*
;
Myoclonus*
;
Stents*