No abstract available.
Cerebral Hemorrhage*

No abstract available.
Cerebral Hemorrhage*

No abstract available.
Cerebral Hemorrhage*

We present a 66-year-old male presenting with sudden onset of headache. Medical decompression was done and neuroimaging of plain cranial CT angiogram (CTA) and CT venogram (CTV) showed components of subarachnoid hemorrhage, intracerebral hemorrhage and hyperdense appearance of the superior sagittal sinus and proximal left transverse sinus. The following were done to look for etiologic factors: 1. fungal swab to determine the cause of the intracerebral hemorrhage secondary to venous thrombosis as the patient presented with a chronic history of sinusitis, 2. coagulopathy workup to look for a hypercoagulable state, and 3. workup for systemic disease of autoimmune in etiology. Anticoagulation was safely initiated within several days given the regression of the intracerebral hemorrhage along with stable findings of subarachnoid hemorrhage. Here we report cerebral venous sinus thrombosis rarely presenting as acute subarachnoid hemorrhage and intracerebral hemorrhage.
Subarachnoid Hemorrhage ; Cerebral Hemorrhage

Objectives: Introduction of the operative technique, creteria of indication for the operation and results. Subjects: 75 patients in 2000 (male: 56; female: 19) The results: the cerebral haemorrhage due to cerebro-vascular accident sometimes were intervented by surgery (specific indication) which helped improving the internal treatment. The procedure of operation involved the drilling the skull and aspiration of the blood. The creteria for surgery by experiences of 100 operations were no limitation of age, Glasgow score: 6; diameter of the blood aggregation : 2 cm. Position of the blood aggregation: any position excluding the cerebral stem. The internal treatment was continued and rehabitation (if having paralysis).
Hemorrhage ; Cerebral Arterial Diseases ; therapeutics ; Cerebral Hemorrhage

In a study in 90 patients with primary hypertensive intracerebral hemorrhage in Friendship Hospital from August 1998 to May 2000. We found that white blood cell were 64.5% when the patients admitted to hospital. The increase of cholesterol is 53.4%; triglycerid is 63.3%. This is rich factor of primary hypertensive intracerebral hemorrhage. The author suggested the above mentioned clinic signs give the early diagnose, fast indication and blood test.
Cerebral Hemorrhage ; Hypertension ; Cerebral Hemorrhage, Traumatic

No abstract available.
Cerebral Hemorrhage* ; Mucormycosis*

No abstract available.
Cerebral Hemorrhage* ; Tuberous Sclerosis*

No abstract available.
Cerebral Hemorrhage ; Parkinson Disease

No abstract available.
Cerebral Hemorrhage* ; Pathology*