No abstract available.
Fetal Death*

No abstract available.
Fetal Death*

No abstract available.
Fetal Death*

No abstract available.
Chickenpox* ; Fetal Death* ; Pregnancy*

No abstract available.
Constriction, Pathologic* ; Fetal Death*

No abstract available.
Fetal Death* ; Umbilical Cord*

No abstract available.
Fetal Death* ; Gastroschisis*

Cystic masses are among the most common sonographically detectable abnormalities of the umbilical cord. Umbilical cord cysts have been described in association with fetal anomalies, with chromosomal or structural defects being found in over 20% of cases. We present a case of umbilical cord cyst associated with fetal death at 29 weeks of gestation with a brief review of literatures.
Fetal Death* ; Pregnancy ; Umbilical Cord*

Fetal growth restriction (FGR) is characterized by fetal compromise and delayed neurological maturation. We report 3 cases of early FGR in the 26th week of gestation, based on hemodynamic Doppler monitoring, conventional cardiotocography, and non-invasive fetal electrocardiography (NI-FECG). Fetal heart rate variability (HRV), beat-to-beat variations, and fetal autonomic brain age scores (fABASs) were normal despite the absence of umbilical diastolic flow in the first case and the pregnancy continued to 30 weeks. NI-FECG helped achieve better fetal maturity. Fetal HRV and fABASs were low in the second and third cases. Fetal demise occurred soon in both cases. We conclude that NI-FECG could be a prospective method for the detection of fetal distress in early FGR.
Brain ; Cardiotocography ; Electrocardiography ; Embryonic and Fetal Development ; Female ; Fetal Death ; Fetal Development ; Fetal Distress ; Fetus ; Heart Rate, Fetal ; Hemodynamics ; Methods ; Pregnancy ; Prospective Studies

The effect of the death or removal of the fetuses at the middle and late stages of pregnancy to the placenta in rats is studied. Either death or removal of the fetuses during pregnancy brought rapid degeneration of all elements of the placenta, first in the yolk sac followed by labyrinth and junctional zone leading into ischemic necrosis secondary to the thrombosis of the maternal blood vessels supplying the placenta. Trophoblasts in the junctional zone survived for a short period of time, but no evidence of abnormal or continuous proliferation of them was noted. Shortly after the death or removal of fetuses cystic degeneration in the junctional zone was observed, but no definite evidence of mole like change was noted. Findings of periodic acid Schiff's reaction, methyl-green pyronine staining and Feulgen reaction in the normal and degenerating placentas are described. The circulatory factors and structural differences between the rat and human placentas are discussed to account for differences observed in the placental changes following the death of fetuses in rats from those of human.
Animals ; Female ; Fetal Death/*pathology ; Placenta/*pathology ; Pregnancy ; Rats