1.Apoptosis and Bcl-2 in Astrocytic Tumors.
Yeon Gyoe JANG ; Kum WHANG ; Soon Won HONG
Journal of Korean Neurosurgical Society 2000;29(4):486-490
No abstract available.
Apoptosis*
2.Bcl-2 Expression of the Primary Cutaneous Malignancies.
Sam Hyeong KIM ; Hoon KANG ; Sook Ja SON
Korean Journal of Dermatology 1997;35(5):847-853
No abstract available.
Apoptosis
3.Molecular Biological Approach to Apoptosis in the Developing Lung.
Journal of the Korean Pediatric Society 2001;44(3):246-251
No abstract available.
Apoptosis*
;
Lung*
4.Apoptosis and Nuclear Shapes in Benign Prosta Hyperplasia and ProstateAdenocarcinoma: Comparsion and Relation.
Jang Wook SONG ; Nak Gyeu CHOI
Korean Journal of Urology 2000;41(2):317-322
No abstract available.
Apoptosis*
;
Hyperplasia*
5.Apoptosis of bone cells and bisphosphonates.
Korean Journal of Medicine 1999;57(4):472-476
No abstract available.
Apoptosis*
;
Diphosphonates*
6.Some discussion of apoptosis.
Journal of Medical Research 1998;7(3):34-35
Like every biological body, the cells under 4 phases: generation growth, development and end. There are 2 ways of the end of cell: necrosis death or apoptosis. The apoptosis related with some diseases that these disorders may be etiological mechanism of diseases. Today, the etiological mechenism of apoptosis has been discovered. The ways of inhibition or stimulation of this process in diseases of increased apoptosis are being studied.
Apoptosis
;
cells
7.Cellular Senes cence, Apoptosis and Carcinogenesis.
Journal of the Korean Geriatrics Society 2004;8(1):1-7
No abstract available.
Apoptosis*
;
Carcinogenesis*
8.Apoptosis of Chondro cytes and Its Regulation in Pathogenesis of Osteoarthritis.
Hyon Jeong KIM ; Eui Cheol SHIN ; Nam Jin YOO ; Jeon Han PARK ; Sung Jae KIM ; Eung Shick KANG ; Jun Seop JAHNG
The Journal of the Korean Rheumatism Association 2001;8(2):104-117
No abstract available.
Apoptosis*
;
Osteoarthritis*
9.Mitochondrial therapy: a new strategy for treating mitochondrion-associated diseases.
Chinese Journal of Biotechnology 2021;37(4):1168-1177
Mitochondrion is a multifunctional organelle in cells and responsible for energy production, cell apoptosis and various life processes. Dysfunctional mitochondria are associated with hundreds of diseases. Increasing evidences have shown that extracellular mitochondria can be endocytosed by cells, directly into cells, and then play roles in cells. Mitochondria are the organelles that are extremely sensitive to oxygen content and pH of microenvironment that induces the adverse effect based on the cellular environment: mitochondria will increase cell survival and viability when they arrive in cells of physiological environment, but mitochondria will cause cell death when they enter the hypoxic and acidic tumor tissues, because they can produce a large amounts of oxygen free radicals. The pharmacological feature of environmental responsiveness of mitochondria could make them as a potential biological drug to kill cancer cells and restore the function of damaged tissues. Currently, mitochondria are used in the treatment of central nervous system diseases (Parkinson's disease, depression, schizophrenia, etc.), peripheral system diseases (ischemic myocardial injury, fatty liver, emphysema, etc.) and tumor. In this review, we summarize the research progress, medical application and challenges of mitochondrial therapy.
Apoptosis
;
Mitochondria
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