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Volume: 34 Issue: 1

1. Recent Progress in Understanding the Mechanisms of Pain and Itch: the Second Special Issue. Page:1—3
2. Differential Inhibition of Nav1.7 and Neuropathic Pain by Hybridoma-Produced and Recombinant Monoclonal Antibodies that Target Nav1.7 : Differential activities of Nav1.7-targeting monoclonal antibodies. Page:22—41
3. Regulation of Pain and Itch by TRP Channels. Page:120—142
4. Combining Human and Rodent Genetics to Identify New Analgesics. Page:143—155
5. Modeling Pain Using fMRI: From Regions to Biomarkers. Page:208—215
6. Involvement of NF-κB and the CX3CR1 Signaling Network in Mechanical Allodynia Induced by Tetanic Sciatic Stimulation. Page:64—73
7. Somatosensory Neuron Typing with High-Coverage Single-Cell RNA Sequencing and Functional Analysis. Page:200—207
8. Sex-Dependent Glial Signaling in Pathological Pain: Distinct Roles of Spinal Microglia and Astrocytes. Page:98—108
9. Inflammatory Changes in Paravertebral Sympathetic Ganglia in Two Rat Pain Models. Page:85—97
10. Investigation of Pain Mechanisms by Calcium Imaging Approaches. Page:194—199
11. Empathy for Distress in Humans and Rodents. Page:216—236
12. Spinal Circuits Transmitting Mechanical Pain and Itch. Page:186—193
13. Cortical Representation of Pain and Touch: Evidence from Combined Functional Neuroimaging and Electrophysiology in Non-human Primates. Page:165—177
14. Expression and Role of Voltage-Gated Sodium Channels in Human Dorsal Root Ganglion Neurons with Special Focus on Nav1.7, Species Differences, and Regulation by Paclitaxel. Page:4—12
15. Proteomic Analysis of the Hippocampus in Mouse Models of Trigeminal Neuralgia and Inescapable Shock-Induced Depression. Page:74—84
16. Chemokine Receptor CXCR3 in the Spinal Cord Contributes to Chronic Itch in Mice. Page:54—63
17. Modulation of Pain and Itch by Spinal Glia. Page:178—185
18. Spinal Mechanisms of Itch Transmission. Page:156—164
19. TNF-α/TNFR1 Signaling is Required for the Full Expression of Acute and Chronic Itch in Mice via Peripheral and Central Mechanisms. Page:42—53
20. Spinal CCL2 Promotes Central Sensitization, Long-Term Potentiation, and Inflammatory Pain via CCR2: Further Insights into Molecular, Synaptic, and Cellular Mechanisms. Page:13—21
21. Infection, Pain, and Itch. Page:109—119