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WPRIM Management System>
DCMS
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Chinese Journal of Burns
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2005
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21
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6
Volume:
21
Issue:
6
1. Morphological changes in intestinal villi after severe burns in rats.
Page:459—461
2. The influence of non-peptide NK1 receptor antagonist L-703, 606 on the early tissue edema formation in rats with deep partial-thickness scald.
Page:455—458
3. Expression of thrombospondin 2 during the repair process after alkali burn injury of cornea in mice.
Page:452—454
4. An experimental study on the role of protein kinase C in the down-regulation of fibroblast proliferation in normal skin and hyperplastic scar by adrenaline.
Page:448—451
5. The influence of topical application of insulin on the formation of basement membrane in scalded rats.
Page:445—447
6. The protective effect of high density lipoprotein on the cardiac function of rats with severe burns.
Page:442—444
7. Biological characteristics of human keratinocytes cultured in serum-free medium without feeder layer.
Page:438—441
8. Influence of nuclear factor-kappaB activation on the expression of cytokines in monocytes stimulated by burn serum.
Page:434—437
9. Influence of polysaccharide from Aloe vera on the proliferation of the human epithelial cells cultured in vitro.
Page:430—433
10. Activation of p38 MAPK signal transduction pathway by burn serum and the expression of VCAM-1 in HUVECs induced by NF-kappaB.
Page:426—429
11. Influence of glucose-insulin-potassium on the levels of inflammatory cytokines and prognosis of MODS in the scalded rats.
Page:422—425
12. The modulating role of p38 mitogen-activated protein kinase in the expression of tumor necrosis factor-alpha in hepatic cells and its role in hepatic injury in severely burned rats.
Page:418—421
13. The role of Janus kinase-signal transducer and transcription activator pathway in the regulation of synthesis and release of lipopolysaccharide-induced high mobility group box-1 protein.
Page:414—417
14. The role of different subtypes of G protein in the secretory function of macrophages stimulated by activated complement following severe burn injury.
Page:410—413
15. The pathogenetic factors, molecular mechanism and the management strategies of postburn inflammatory reaction.
Page:405—409
16. Status in quo of postburn immune study.
Page:401—404