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WPRIM Management System>
DCMS
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Chinese Journal of Industrial Hygiene and Occupational Diseases
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2008
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26
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1
Volume:
26
Issue:
1
1. Treatment of coal worker pneumoconiosis associated with pulmonary heart disease and hyposmolarity.
Page:64—64
2. A case of asbestos induced malignant mesothelioma.
Page:63—63
3. Determination, analysis and evaluation system for dusts in work places in Germany.
Page:60—62
4. Monitoring for silicon dust hazards in USA and analysis of epidemiological data of exposed population.
Page:58—60
5. Control and management of dusts hazards in USA.
Page:56—58
6. Brief introduction of selection and application of respiratory protection articles for occupational populations in USA.
Page:54—55
7. Serum resistin and leptin level in patients with coal worker pneumoconiosis and clinical significance.
Page:52—53
8. Survey on patients infected with extended-spectrum beta-lactamases producing bacteria.
Page:50—51
9. Influential factors for amount of residual fluid in whole lung lavage and treatment.
Page:48—50
10. Sinus tachycardia after whole lung lavage in 50 patients.
Page:47—48
11. Survey on occupational hazards of antimony oxide dusts.
Page:44—46
12. Analysis for characteristics of identified pesticides in five years in a province.
Page:43—44
13. Clinical study on pneumoconiosis with pulmonary thromboembolism.
Page:39—42
14. Value of tumor markers series of hydrothorax in differential diagnosis of pleural effusion.
Page:34—38
15. Late-onset silicosis in 15 patients.
Page:33—33
16. A retrospective cohort study on mortality among silicotic workers in Hong Kong with emphasis on lung cancer.
Page:29—33
17. Mortality analyses for tungsten miners.
Page:24—28
18. Relationship between aryl hydrocarbon receptor G1661A gene polymorphism and level of urinary 1-hydroxypyrene of coke oven workers.
Page:20—23
19. Relationship between the expression of heat shock protein and genetic damage in peripheral blood of workers exposed to coke oven emissions.
Page:16—19
20. Role of activator protein-1 in pathogenesis of silicosis: an in-vitro study.
Page:12—15
21. Polymorphisms of FAS and FASL genes and susceptibility of silicosis.
Page:7—11
22. ERK, JNK/AP-1 pathway was involved in silica-induced cell cycle changes.
Page:3—6
23. Achievements of research on pathogenicity of dusts in urgent need of combination of prevention and treatment of dusts induced diseases.
Page:1—2