Clinical features of vestibular syncope associated with tumarkin attacks in delayed endolymphatic hydrops.
10.3760/cma.j.cn115330-20201208-00912
- VernacularTitle:迟发性膜迷路积水患者前庭性晕厥的临床分析
- Author:
Yang Ming LENG
1
;
Ren Hong ZHOU
1
;
Jing Jing LIU
1
;
Hong Chang WANG
1
;
Jian CHEN
2
;
Bo LIU
1
Author Information
1. Department of Otorhinolaryngology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430022, China.
2. Department of Cardiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430022, China.
- Publication Type:Journal Article
- MeSH:
Endolymphatic Hydrops/diagnosis*;
Humans;
Retrospective Studies;
Syncope;
Vestibular Evoked Myogenic Potentials;
Vestibule, Labyrinth
- From:
Chinese Journal of Otorhinolaryngology Head and Neck Surgery
2021;56(11):1194-1198
- CountryChina
- Language:Chinese
-
Abstract:
Objective: To analyze the clinical characteristics of vestibular syncope (VS) associated with drop attacks (DA) in delayed endolymphatic hydrops (DEH). Methods: DEH cases with complete data were retrospectively analyzed, including three DEH cases with DA and VS (VS group), and six DEH cases without DA or VS (control group). The clinical profile, the results of neurotological examinations [such as pure tone audiometry, electrocochleography (EcochG), caloric test, vestibular evoked myogenic potentials (VEMP), and video head impulse test (vHIT)] and treatment outcomes were analyzed. Results: (1) In the VS group, there were three cases of ipsilateral DEH; in the control group, there were six cases of ipsilateral type. One case in each group had a history of migraine. (2) The prevalence of abnormal results in caloric test, vHIT, cervical VEMP, and ocular VEMP in the VS group was 3/3, 1/3, 2/2, and 2/2, respectively, and in the control group was 3/6, 0/3, 1/6, and 4/6, respectively. Two cases in each group underwent EcochG, and no identifiable waveform was elicited on the affected side, and-SP/AP ratio of unaffected side was less than 0.4. (3) Patients in both groups were initially treated with conservative medication. Two cases in the VS group subsequently received intratympanic injections of dexamethasone. No DA or VS occurred during a follow-up period lasting over one year. All patients achieved good control of vertigo during the follow-up period. Conclusions: VS may occur in the patients with DEH. The differential diagnosis of syncope in patients with otogenic vertiginous disease can help improve clinical diagnosis and treatment.
