A Case of Panhypopituitarism with Rhabdomyolysis.
10.3803/jkes.2005.20.2.174
- Author:
Sung Wook HONG
1
;
Eun Jung LEE
;
Ji Young PARK
;
Ji Sung YOON
;
Ji O MOK
;
Yeo Joo KIM
;
Hyeong Kyu PARK
;
Jae Woo KIM
;
Chul Hee KIM
;
Sang Jin KIM
;
Dong Won BYUN
;
Kyo Il SUH
;
Myung Hi YOO
Author Information
1. Department of Internal Medicine College of Medicine, Soonchunhyang University, Chunan, Korea.
- Publication Type:Case Report
- Keywords:
Rhabdomyolysis;
Panhypopituitarism
- MeSH:
Coma;
Creatine Kinase;
Diabetic Ketoacidosis;
Humans;
Hypothyroidism;
Muscle Cells;
Plasma;
Rhabdomyolysis*;
Thyrotoxicosis
- From:Journal of Korean Society of Endocrinology
2005;20(2):174-178
- CountryRepublic of Korea
- Language:Korean
-
Abstract:
Rhabdomyolysis is the consequence of extensive muscle injury with the release of muscle cell constituents into plasma. It can arise from trauma and also from a variety of nontraumatic causes. Trauma, drugs, toxins and infection are the major causes of rhabdomyolysis, but it is rarely associated with metabolic disorders such as severe electrolyte disturbance, diabetic ketoacidosis, hyperosmolar nonketotic coma, hypothyroidism and thyrotoxicosis. There have been several reported cases of metabolic rhabdomyolysis, but panhypopituitarism as a cause has never been identified. We experienced a case of acute rhabdomyolysis associated with panhypopituitarism. Thus, So we report this case with the review of related literature. Metabolic disorder is a rare cause of rhabdomyolysis, but it should always be considered in a patient having and unexplained increased of the creatine kinase concentration
