A Case of Primary Hyperparathyroidism Associated with Proximal Renal Tubular Acidosis and Postoperative Hungry Bone Syndrome
- Author:
Je Ho HAN
;
Kun Ho YOON
;
Bong Yun CHA
;
Ho Young SON
;
Kwang Woo LEE
;
Hae Ok JUNG
;
Chang Sup KIM
;
Moo Il KANG
;
Chul Soo CHO
;
Ho Yun KIM
;
Sung Koo KANG
- Publication Type:Case Report
- Keywords:
Primary hyperparathyroidism;
Parathyroid adenoma;
Hungry bone syndrome
- MeSH:
Acidosis;
Acidosis, Renal Tubular;
Adenoma;
Calcium;
Hyperparathyroidism, Primary;
Hypocalcemia;
Hypophosphatemia;
Kidney Tubules, Proximal;
Metabolism;
Parathyroid Glands;
Parathyroid Hormone;
Parathyroid Neoplasms;
Phosphorus
- From:Journal of Korean Society of Endocrinology
1994;9(2):141-149
- CountryRepublic of Korea
- Language:Korean
-
Abstract:
Primary hyperparathyroidism is a generalezed disorder of calcium, phosphorus and bone metabolism due to an increased secretion of parathyroid hormone. Single parathyroid adenoma is the most common cause of primary hyperparathyroidism. Because parathyroid hormone has been proposed as an important inhibitor of renal bicarbonate reabsorption of proximal tubule, proximal renal tubular acidosis is not rare in primary hyperparaphyroidism. After parathyroid resection, significant hypocalcemia and hypophosphatemia requiring prolonged medical management may develop, termed hungery bone syndrome. We experienced a case of primary hyperparathyroidism associated with proximal renal tubular acidosis, and severe hungry bone syndrome after resection of the adenoma of parathyroid gland.
