Pathophysiology of Vestibular Migraine
- Author:
Sun Young OH
1
;
Tae Ho YANG
Author Information
1. Department of Neurology, Chonbuk National University College of Medicine, Jeonju, Korea. ohsun@jbnu.ac.kr
- Publication Type:Review
- Keywords:
Migraine;
Vertigo;
Pathophysiology
- MeSH:
Calcitonin;
Calcium Channels;
Cerebellar Diseases;
Cortical Spreading Depression;
Dopamine;
Meniere Disease;
Migraine Disorders;
Motion Sickness;
Neurotransmitter Agents;
Norepinephrine;
Psychotic Disorders;
Serotonin;
Vertigo;
Vestibular Neuronitis
- From:Journal of the Korean Balance Society
2012;11(3):81-87
- CountryRepublic of Korea
- Language:Korean
-
Abstract:
Vestibular migraine (VM) is an increasingly recognized cause of episodic recurrent vertigo. However, the pathophysiology of VM is still a matter of speculation. An understanding of the relationship between migraine and the vestibular system increases knowledge of the pathogenesis of both migraine and vertigo. The pathophysiology of VM has been known to be related to cortical spreading depression, neurotransmitters (i.e., serotonin, noradrenaline, dopamine, calcitonin gene-related peptide) and calcium ion channel disorder. Moreover, VM is related with Meniere's disease, benign paroxysmal positional vertigo, motion sickness, cerebellar dysfunction, or comorbid psychotic disorder. This review refines recently proposed pathophysiological concept for VM and relationships between migraine and other related disorders.
