Digital Sympathectomy for Treatment of Raynaud's Syndrome.
- Author:
Se Whan RHEE
1
;
Hee Chang AHN
;
M Seung Suk CHOI
;
Chang Yeon KIM
Author Information
1. Department of Plastic and Reconstructive Surgery, College of Medicine, Hanyang University, Seoul, Korea. ahnhc@hanyang.ac.kr
- Publication Type:Original Article
- Keywords:
Raynaud's syndrome
- MeSH:
Adventitia;
Arteries;
Calcium Channels;
Female;
Fingers;
Hand;
Humans;
Male;
Necrosis;
Spasm;
Sympathectomy*;
Vasodilator Agents;
Weather
- From:Journal of the Korean Society of Plastic and Reconstructive Surgeons
2005;32(4):479-484
- CountryRepublic of Korea
- Language:Korean
-
Abstract:
Raynaud's syndrome causes discolorization, ischemic claudication(pain) and necrosis of the digits through insufficiency in the circulation which is induced by intermittent spasms of the digital arteries. From January, 2002 to December, 2004, 10 patients were surgically treated for Raynaud's syndrome. 9 patients were female and 1 patient was male. 2 patients showed unilateral involvement, 8 patients were operated on both hands. 6 patients had necrotic changes on the finger tips due to the disease. Ages ranged from 21 to 60 with an average of 39.1. Ischemic pain, discolorization, and cold intolerance of the digits were the common symptoms. All patients were evaluated with color doppler before the surgery. Two different procedures were applied according to the severity of the disease: Patients with decreased circulation received, what we call a limited digital sympathectomy, i.e. stripping of the adventitia of the ulnar, radial and common digital arteries. An extended procedure, radical digital sympathectomy, was performed on patients with a complete block of circulation. Stripping of the adventitia in these patients also involved the proper digital arteries. Symptoms like discolorization, ischemic pain, and cold intolerance improved immediately after the surgery. The patients did not suffer from pain even with exposure to cold weather. We conclude that digital sympathectomy could improve the symptoms in Raynaud's patients who do not respond to conservative treatment such as calcium channel blocker and other vasodilators.
