Furosemide-Induced Nephrocalcinosis in Very Low Birth Weight Infants.
- Author:
Mi Jung PARK
;
Kook In PARK
;
Min Soo PARK
;
Ran NAMGUNG
;
Chul LEE
;
Dong Gwan HAN
;
Hyunee YIM
;
Woo Hee JUNG
- Publication Type:Case Report
- MeSH:
Bronchopulmonary Dysplasia;
Cytomegalovirus;
Cytomegalovirus Infections;
Ductus Arteriosus, Patent;
Furosemide;
Heart Failure;
Heart Septal Defects, Ventricular;
Humans;
Hypercalciuria;
Infant*;
Infant, Newborn;
Infant, Premature;
Infant, Very Low Birth Weight*;
Kidney Calculi;
Lung Diseases;
Nephrocalcinosis*;
Pneumonia
- From:Journal of the Korean Pediatric Society
1994;37(4):553-559
- CountryRepublic of Korea
- Language:Korean
-
Abstract:
Renal calcifications are a recognized complication of furosemide therapy in premature infants. Particularly in infants with chronic lung disease, the use of this potent diuretic is associated with hypercalciuria, which may predispose the infant to medullary nephrocalcinosis and renal calculi, We experienced two cases of nephrocalcinosis in very low birth weight infants. One had bronchopulmonary dysplasia, pneumonia, patent ductus arteriosus, ventricular septal defect and congestive heart failure and the other had systemic cytomegalovirus infection with cytomegalovirus pneumonitis and ricket of prematurity. Both patients received a large amount of furosemide. We presented these cases with brief review of literatures.
