Study on Thrombocytic Ultrastructure and Functions of Blood Stasis Syndrome in Liver Disorders.
- VernacularTitle:肝病血瘀证患者血小板超微结构和功能的研究
- Author:
Chizhi ZHANG
- Publication Type:Journal Article
- Keywords:
Hepatic disorders/pathology blood stasis/ pathology platelet/ultrastructure
- From:
Journal of Traditional Chinese Medicine
1992;0(12):-
- CountryChina
- Language:Chinese
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Abstract:
The results of this study revealed that there was less discoid cell and more pseudopodium, more thrombocytes tended to agglutinate with increased proportion of hyper - glycogenic platelets. There were also platelets sticking to RBC. WBC and lymphocytes. The glanules within the platelates were decreased or even fused together, unable to differentiate them. The vascular system were dilated and proliferated. Abnormal granules were seen with its size similar to a - granules, covered with membrane, with cytoplasm and metabolites inside. Under the revulsion of ADP, there was 70. 28% agglutining body. Most intercellular spaces revealed unclear bridge - form connection or even disappeared with fusion of cellular membrane. All these changes were more distinctive than that in the control group, demonstrating the changes in ultrasturcture and agglutination function might be one of the causes of blood stasis in hepatopathy.
