Detection of periodontal disease related bacteria from the implant-abutment interface in oral cavity.
- Author:
Meung Ju HAN
1
;
Chae Heon CHUNG
;
Hee Jung KIM
;
Joong Ki KOOK
;
So Young YOO
Author Information
1. Department of Prosthodontics, College of Dentistry, Chosun University, Gwangju, Korea. jhajung@chosun.ac.kr
- Publication Type:Original Article
- Keywords:
Implant-abutment interface;
Periodontopathogens;
Microbial leakage;
Porphyromonas gingivalis
- MeSH:
Bacteria;
Colon;
DNA, Ribosomal;
Forsythia;
Humans;
Mouth;
Periodontal Diseases;
Polymerase Chain Reaction;
Porphyromonas gingivalis;
Prevotella intermedia;
Tongue;
Treponema denticola
- From:The Journal of Korean Academy of Prosthodontics
2008;46(2):116-124
- CountryRepublic of Korea
- Language:Korean
-
Abstract:
STATEMENT OF PROBLEM: Implant systems result in gaps and cavities between implant and abutment that can act as a trap for bacteria and thus possibly cause inflammatory reactions in the peri-implant soft tissues. PURPOSE: Porphyromonas gingivalis, Prevotella intermedia, Tannerella forsythia, Treponema denticola, and Aggregatibacter actinomycetemcomitans, related to implant-abutment interface microleakage. MATERIAL AND METHODS: Samples were taken from 27 subjects with sterilized paper points and were transported in 1XPBS. The detection of periodontopathogens were performed by polymerase chain reaction with species-specific primers based on 16S rDNA. RESULTS: Our data showed that the detection rate of P. gingivalis and P. intermedia in implant fixture was 59% and 82% in patients respectively. Detection rate of P. gingivalis and P. intermedia in implant crevice was 44% and 82% in patients. Detection rate of P. gingivalis and P. intermedias in tongue was 82% and 82% in patients. CONCLUSION: Current implant systems cannot safely prevent microbial leakage and bacterial colonization of the inner part of the implant.
