Apraxia of Lid Opening after Traumatic Brain Injury: A Delayed Complication.
- Author:
Sung Gyun JOO
1
;
Mee Young PARK
;
Jun LEE
;
Yong Kook CHO
;
Jung Hyun KIM
;
Ji Yeun CHUNG
Author Information
1. Department of Neurology, Yeungnam University College of Medicine, Daegu, Korea. mypark@med.yu.ac.kr
- Publication Type:Case Report
- Keywords:
Apraxia of lid opening;
Traumatic brain injury;
Non dominant frontal lobe
- MeSH:
Apraxias*;
Brain;
Brain Injuries*;
Encephalomalacia;
Frontal Lobe;
Humans;
Magnetic Resonance Imaging;
Tomography, X-Ray Computed
- From:Journal of the Korean Neurological Association
2005;23(5):682-685
- CountryRepublic of Korea
- Language:Korean
-
Abstract:
Apraxia of lid opening (ALO) has been suggested to be a dysfunction of the supranuclear control of the levator palpebrae superioris caused mainly by basal ganglial lesion. The hypometabolism of the medial frontal lobe may be a pathophysiologic mechanism in ALO. We report two ALO patients who developed these symptoms as a delayed complication after traumatic brain injury (TBI). Their MRI showed encephalomalacia in the Rt. medial frontal cortex, which was not shown in initial brain CT scans. Delayed pathologic changes after TBI may contribute to the development of ALO in these cases.
