Hemodynamic Profile of Carotid Sinus Nerve Blockade on Hypotension Occurred Immediately after Carotid Endarterectomy Declamping: A case report.
10.4097/kjae.2006.51.5.651
- Author:
Su Jin KANG
1
;
Won Jung SHIN
;
Gyu Sam HWANG
Author Information
1. Department of Anesthesiology and Pain Medicine, Asan Medical Center, University of Ulsan College of Medicine, Seoul, Korea. kshwang@amc.seoul.kr
- Publication Type:Case Report
- Keywords:
carotid sinus nerve blockade;
hemodynamic profile
- MeSH:
Arterial Pressure;
Blood Pressure;
Cardiac Output;
Carotid Sinus*;
Endarterectomy;
Endarterectomy, Carotid*;
Hemodynamics*;
Hypotension*;
Nerve Block*;
Retrospective Studies;
Stroke Volume;
Vascular Resistance
- From:Korean Journal of Anesthesiology
2006;51(5):651-654
- CountryRepublic of Korea
- Language:Korean
-
Abstract:
Carotid sinus nerve blockade (CSNB) has been shown to be effective for management of post-carotid endarterectomy (CEA) hypotension. The underlying hemodynamic changes are unknown. We retrospectively analyzed a recorded arterial blood pressure (ABP) waveform using a Modelflow technique. After declamping, total peripheral resistance (TPR) decreased (-65% of predeclamping value), but cardiac output (CO) and stroke volume (SV) increased (+60 and +57% of predeclamping value, respectively). CSNB abruptly increased blood pressure (BP) and TPR (75 and 95% of nadir, respectively), while CO and SV gradually decreased to -21 and -16% of maximum value, respectively). In conclusion, we found that severe decline of TPR was a cause of severe hypotension after declamping and CSNB increased BP mainly through increase in TPR, with little change in CO and SV.
