Effects of Compression Rate and Compression/Relaxation Ratio on the Hemodynamics of Simultaneous Sterno-Thoracic Cardiopulmonary Resuscitation in the Canine Model of Cardiac Arrest.
- Author:
Seo Young LEE
1
;
Sung Oh HWANG
;
Hyun KIM
;
Yong Soo JANG
;
Han Joo CHOI
;
Sung Bum OH
;
Kyoung Chul CHA
;
Kang Hyun LEE
;
Jung Han YOON
;
Byung Su YOO
;
Seung Hwan LEE
;
Kyung Hoon CHOE
Author Information
1. Department of Emergency Medicine, Wonju Medical College, Yonsei University, 162 Ilsandong, Wonju, Gangwondo, Korea. shwang@wonju.yonsei.ac.kr
- Publication Type:Original Article
- Keywords:
Cardiopulmonary resuscitation;
Cardiac arrest
- MeSH:
Animals;
Cardiopulmonary Resuscitation*;
Dogs;
Heart Arrest*;
Hemodynamics*;
Relaxation;
Ventricular Fibrillation
- From:Journal of the Korean Society of Emergency Medicine
2003;14(5):522-528
- CountryRepublic of Korea
- Language:Korean
-
Abstract:
PURPOSE: This study was designed to determine the compression rate and the compression/relaxation ratio to produce the optimal hemodynamic effect with simultaneous sterno-thoracic cardiopulmonary resuscitation (SST-CPR)and to investigate the mechanism for the blood flow generated by SST-CPR. METHODS: A canine model of ventricular fibrillation was used. Twelve mongrel dogs were divided into two groups. In the first six animals were resuscitated by using SST-CPR with the compression duration varied randomly at 2-minute intervals, 30%, 40%, and 50% of the CPR cycle, at a constant rate of 80/min. In the other six dogs, SST-CPR was performed with a randomly varied compression rate, 60, 80, 100, and 120/minute, at a 50:50 compression/relaxation ratio. RESULTS: During SST-CPR, increasing the compression relaxation ratio from 30:70 to 50:50 increased the end tidal CO2 from 10+/-2 mmHg to 15+/-3 mmHg. Increasing the compression rate from 60 to 100/minute tended to improve the carotid blood flow. CONCLUSION: The maximal hemodynamic effects with SSTCPR was generated when the compression rate was 100/minute and the compression/relaxation ratio was 50:50. A combination of the cardiac and the thoracic pump theories may be the mechanism for the blood flow produced by SST-CPR.
