A Case of Anetoderma of Jadassohn-Pellizali.
- Author:
See Yong PARK
;
Choong Rim HAW
;
Soo Nam KIM
;
Tae Yun YOO
- Publication Type:Original Article
- MeSH:
Adult;
Anetoderma*;
Atrophy;
Dermis;
Diagnosis;
Elastic Tissue;
Fingers;
Humans;
Male;
Numismatics;
Shoulder;
Skin;
Subcutaneous Tissue;
Upper Extremity
- From:Korean Journal of Dermatology
1977;15(3):347-351
- CountryRepublic of Korea
- Language:Korean
-
Abstract:
Anetoderma or macular atrophies is a localized laxity of the skin, resulting from defect in the elastie tissue of the dermis. This defect produces a clinical picture consisting of circumscribed area of soft, thin and wrinkled skin, which can be pushed easily into the subcutaneous tissue by the examining finger. When the lesions appear without any preceding recognizable disorder, they considered to be primary macular atrophies. In this group, based on whether an inflammatory reaction has occurred before the appearance of macular atrophy, two types aer generally acknowledged: Anetoderma of Jadassohn-Pellizali has a preceding inflammatory reaction, whereas anetoderma of Schweninger-Buzzi has had no preceding discemible inflammatory changes. The authors observed a case of anetoderma of Jadassohn-Pellizali in a 34 year old male when showed bean to coin sized rovnd or oval, thin, soft and wrinkled macules on the trunk, shoulder and upper extremities. About 3 years ago, erythematous macules developed which extended for several months to reach a size of l.5 to 2.5 cm. Slowly each lesion faded from the cener to outward Ieaving soft and wrinkled macules(Fig.l). Routine physical and laboratory findings were within-normal-limit. Histopathological findings showed scanty and fragmentation of the elastic fiber of the dermis and perivascular non-specific inflammatory cell infiltration. (Fig. 2, 3 & 4) Diagnosis was confirmed by clinical and histopathological findings. Literature was briefly reviewed for the discussion,
