Clinical Study on the Effect of Intravenous Fluid to the BIood Pressure Change During Spinal Anesthesia .
- Author:
He Sun SONG
1
;
Se Ung CHON
Author Information
- Publication Type:Original Article
- MeSH: Anesthesia, Spinal*; Blood Pressure; Blood Volume; Cardiac Output; Glucose; Heart Rate; Hypotension; Paralysis; Phenoxybenzamine; Vasodilator Agents
- From:Korean Journal of Anesthesiology 1972;5(1):19-23
- CountryRepublic of Korea
- Language:Korean
- Abstract: Hypotension immediately after spinal anesthesia has been considered as the result of pre-ganglionic sympathetic paralysis and secondarily due to changes in cardiac output. And also the increase of vascular bed space about 20~25% was pointed ont during spinal anesthesia which was another cause of spinal hypotension. Same degree of increased vascular bed space was produced by vasodilators such as Dibenzyline and Arfonad. Therefore, vasopressors have been used as the drug of choice to prevent and treat this spinal hypotension. This study was attempted to combat the hypotension during spinal anesthesia by filling this increased vascular bed space with Lactated-Ringer's or Dextrose solution instead of giving vasopressors. Lactated-Ringer's or Dextrose solution was administered to 141 cases, before and immediately after. spinal anesthesia was performed. The given amount of fluid was about 12.5~15% of estimated total blood volume (Group A). And no intravenous fluid was given to 101 cases as control (Group B). Changes of the systolic, diastolic blood pressure and pulse rate were observed. In the Group A, significant changes of the systolic (t=2.52, p<0.02) and diastolic blood pressure (t=1.98, p<0. 05) was observed. The changes of pulse rate were found not significant in the both group series.
