A Case of Stasis Dermatitis with Secondary Lipodermatosclerosis in a Patient with Antithrombin III Deficiency.
- Author:
Jeong Deuk LEE
1
;
Eun Young BAE
;
Chung Eui YOU
;
Sang Hyun CHO
Author Information
1. Department of Dermatology, College of Medicine, The Catholic University of Korea, Seoul, Korea. drchosh@hotmail.com
- Publication Type:Case Report
- Keywords:
Antithrombin III deficiency;
Lipodermatosclerosis;
Stasis dermatitis
- MeSH:
Anticoagulants;
Antithrombin III Deficiency*;
Antithrombin III*;
Dermatitis*;
Fibrin;
Humans;
Hyperpigmentation;
Leg;
Skin;
Venous Insufficiency;
Venous Thromboembolism;
Venous Thrombosis
- From:Korean Journal of Dermatology
2003;41(5):645-648
- CountryRepublic of Korea
- Language:Korean
-
Abstract:
Antithrombin is one of the main endogenous anticoagulants. Antithrombin deficiency may result from hereditary or acquired factors. Inherited antithrombin deficiency is an uncommon autosomal disorder associated with a tendency to venous thromboembolism. Stasis dermatitis occurs as a result of venous stasis caused by venous incompetence or deep vein thrombosis. Furthermore, lipodermatosclerosis that refers to the skin induration and hyperpigmentation of the legs, often occurs in patients who have venous insufficiency. We report a case of stasis dermatitis, complicated by lipodermatosclerosis on both legs of a patient with hereditary antithrombin III deficiency.
