Hypokalemia-Induced Rhabdomyolysis by Primary Aldosteronism Coexistent With Sporadic Inclusion Body Myositis.
10.5535/arm.2015.39.5.826
- Author:
Jong Ha LEE
1
;
Eunkuk KIM
;
Suk CHON
Author Information
1. Department of Physical Medicine and Rehabilitation, Kyung Hee University School of Medicine, Seoul, Korea.
- Publication Type:Case Report
- Keywords:
Rhabdomyolysis;
Aldosteronism;
Hypokalemia;
Inclusion body myositis
- MeSH:
Adenoma;
Adrenalectomy;
Asian Continental Ancestry Group;
Biopsy;
Creatine Kinase;
Humans;
Hyperaldosteronism*;
Hypokalemia;
Lower Extremity;
Male;
Middle Aged;
Muscle Weakness;
Myositis, Inclusion Body*;
Rhabdomyolysis*
- From:Annals of Rehabilitation Medicine
2015;39(5):826-832
- CountryRepublic of Korea
- Language:English
-
Abstract:
We describes a patient with hypokalemia-induced rhabdomyolysis due to primary aldosteronism (PA), who suffered from slowly progressive muscle weakness after laparoscopic adrenalectomy, and was later diagnosed with coexisting sporadic inclusion body myositis (sIBM). A 54-year-old Asian male presented with severe muscle weakness of both lower extremities. Laboratory findings showed profound hypokalemia, and extreme elevation of the serum creatine phosphokinase levels, suggestive of hypokalemia-induced rhabdomyolysis. Further evaluation strongly suggested PA by an aldosterone-producing adenoma, which was successfully removed surgically. However, muscle weakness slowly progressed one year after the operation and a muscle biopsy demonstrated findings consistent with sIBM. This case is the first report of hypokalemia-induced rhabdomyolysis by PA coexistent with sIBM, to the best of our knowledge.
