Creabellar Infarction: A Clinicoradiologic Correlation of 27 Cases.
- Author:
Sang Gull CHO
1
;
Gun Sei OH
;
Jang Je CHUNG
;
Mu Young AHN
;
Hyun Gil SHIN
;
Kwang Ho LEE
;
Dae Ho KIM
Author Information
1. Department of Neurology, College of Medicine, Soonchunghyang University, Korea.
- Publication Type:Original Article
- MeSH:
Arteries;
Ataxia;
Basilar Artery;
Brain;
Brain Stem;
Brain Stem Infarctions;
Cerebellar Ataxia;
Consciousness;
Dizziness;
Facial Paralysis;
Headache;
Hearing Loss;
Humans;
Infarction*;
Intracranial Embolism;
Lateral Medullary Syndrome;
Magnetic Resonance Imaging;
Nausea;
Ocular Motility Disorders;
Pica;
Stroke;
Vertigo;
Vomiting
- From:Journal of the Korean Neurological Association
1993;11(2):164-174
- CountryRepublic of Korea
- Language:Korean
-
Abstract:
We reviewed 27 patients wlth cerebellar infarction which was demonstrated by brain CT and/or MRI. Infarction occurred in the territory of posterior inferior cerebellar artery (PICAj in 16 patients, and the territory of the superior cerebellar artery(SCA) was involved in 5 patients. Antenor inferior cerebellar artery(AICA) infarcts occurred in 3 patients. Both PICA and SCA temtories were involved in 2 patients. In the remaining 1 patient, the infarct encompassed the borderzone between the SCA and PICA territories. The main symptoms and signs were sudden onset of vertigo, dizziness, nausea, vomiting, dysmetria, ataxia, nystagmus, and headache. There were signs of associated brain stem infarction or occipitotemporal infarction; rostral basilar artery syndrome, classic SCA syndrome, Wallenberg syndrome, internuclear ophthalmoplegia, facial palsy, hearing impairment. Presumed cerebral embolism was the main stroke mechanism in the SCA terntories. Six patients with brainstem compression or brainstem involvement showed consciousness deterioration, and only one of them died as a result of extensive cerebellar infarctions involving both SCA and PICA territories Cerebellar infarction may run a more benign course than previously thought.
