Treating secondary organizing pneumonia after infection based on Sanjiao membranous tube theory
10.3969/j.issn.1006-2157.2025.06.002
- VernacularTitle:运用三焦膜性管道论探讨感染后继发机化性肺炎的治疗
- Author:
Yanni LI
1
,
2
;
Xiaomei ZHANG
3
;
Tianshu YANG
1
;
Yunlong SUN
1
;
Mengqian LI
1
;
Yuxin LAI
1
;
Liangduo JIANG
4
Author Information
1. Beijing University of Chinese Medicine
2. Ningxia Hui Autonomous Region Hospital / Research Institute of Traditional Chinese Medicine
3. Dongfang Hospital, Beijing University of Chinese Medicine
4. Dongzhimen Hospital, Beijing University of Chinese Medicine
- Publication Type:Journal Article
- Keywords:
Sanjiao membranous tube theory;
secondary organizing pneumonia after infection;
latent pathogen;
pulmonary micro-membrane;
regulating and dredging the pulmonary micro-membrane
- From:
Journal of Beijing University of Traditional Chinese Medicine
2025;48(6):746-750
- CountryChina
- Language:Chinese
-
Abstract:
Secondary organizing pneumonia after infection is a pathological condition characterized by connective tissue filling and obstructing the alveoli and bronchioles, in which following an infection in the lung, the inflammatory response is not controlled in a timely and effective manner. The pathogenesis and treatment of this condition can be interpreted through the Sanjiao membranous tube theory and the concept of stagnation within the pulmonary micro-membrane. Sanjiao is conceptualized as a four-way membranous tube that internally connects with the zangfu organs and externally with the skin and muscles, enabling the circulation of energy and fluids throughout the body. It also maintains communication with the zangfu micro-membranes. Within the lungs, the pulmonary micro-membrane is distributed and connected to the upper jiao membranous tube, facilitating the movement of qi and fluids and supporting nutrient distribution. External pathogens may invade the Sanjiao membranous system through the external membranous tube, travel internally along this system, and transform into latent pathogens that settle within the pulmonary micro-membrane. These latent pathogens can subsequently transform into heat or dampness, leading to the depletion of lung qi and impairing the lung′s ability to regulate and transport body fluids. Consequently, fluids may seep into the pulmonary micro-membrane, where they are transformed into dampness, turbidity, and phlegm. The accumulation of damp-turbidity and phlegm obstructs the flow of qi and blood, resulting in blood stasis in the pulmonary collaterals. This stagnation occurring within both the pulmonary micro-membrane and its associated collaterals underlies the development of secondary organizing pneumonia after infection. In severe cases, this condition may progress to pulmonary interstitial fibrosis. The therapeutic approach emphasizes expelling latent pathogens, regulating and dredging the pulmonary micro-membrane, tonifying the healthy qi, and supporting health. Regulating and dredging the pulmonary micro-membrane is a crucial step, with a focus on promoting the flow of lung qi, resolving dampness and phlegm, and activating blood circulation to remove stasis.
