A Case of Athetosis in Basal Ganglia Calcification Associated with Hypoparathyroidism.
- Author:
Yoo Mi PARK
1
;
Hye Young KWON
;
Hoo Won KIM
;
Dong Kwon KIM
Author Information
1. Department of Neurology, Taejon, Eulji Hospital, Korea.
- Publication Type:Brief Communication
- MeSH:
Anoxia;
Athetosis*;
Basal Ganglia*;
Carbon;
Cefonicid;
Encephalitis;
Extrapyramidal Tracts;
Glycosaminoglycans;
Humans;
Hypoparathyroidism*;
Hypothyroidism;
Incidence;
Korea;
Nervous System Diseases;
Neurons;
Toxoplasmosis;
Tuberous Sclerosis
- From:Journal of the Korean Neurological Association
1995;13(4):1017-1019
- CountryRepublic of Korea
- Language:Korean
-
Abstract:
Extrapyramidal tract motor disorder in calcification of basal ganglia probably occurs when the deposition of acid mucopolysaccharides in the basal ganglia is severe enough to lead to neuronal loss. Basal ganglia calcification has been noted to occur with higher incidence and intensity in encephalitis lethargica, carbon monocide intoxication, anoxia, tuberous sclerosis, toxoplasmosis, hypothyroidism and hypoparathyroidism. The neurologic disorder is frequently reversible with treatment in patients with basal ganglia calcification who have hypoparathy-roidism. We report a patient with idiopathic hypoparathyroidism associated with bilateral calcification of the basal ganglia and athetoid movement. This is the first case report in Korea.
