A Case of Pseudohyperphosphatemia in a Patient with Multiple Myeloma.
- Author:
Sun Ah LEE
1
;
Jung Lim LEE
;
Kun Woo PARK
;
Hyeong Seok KIM
;
Soo Hyun BAE
;
Soon Hee CHANG
Author Information
1. Department of Internal Medicine, Daegu Fatima Hospital, Daegu, Korea. mozarteffect@hanmail.net
- Publication Type:Case Report
- Keywords:
Multiple myeloma;
Hyperphosphatemia
- MeSH:
Extracellular Fluid;
Humans;
Hypercalcemia;
Hyperphosphatemia;
Hypoparathyroidism;
Light;
Molybdenum;
Multiple Myeloma;
Phosphoric Acids;
Renal Insufficiency;
Uric Acid
- From:Korean Journal of Medicine
2013;84(1):130-134
- CountryRepublic of Korea
- Language:Korean
-
Abstract:
Hyperphosphatemia develops when there is impaired renal phosphate excretion or massive extracellular fluid phosphate load. For example, renal insufficiency, hypoparathyroidism, exogenous phosphate administration, and extensive cellular injury induce a hyperphosphatemic state. In patients with multiple myeloma, renal insufficiency occurs as a result of hypercalcemia, light chain tubulopathy, urate nephropathy or infection, and hyperphosphatemia usually results from renal failure. We report here a case of a patient with multiple myeloma who had an elevated serum phosphate level measured by the phosphomolybdate UV method without significant renal insufficiency and was finally diagnosed with pseudohyperphosphatemia.
