Study on the correlation between the location and early neurological deterioration in isolated subcortical infarction
10.19845/j.cnki.zfysjjbzz.2022.0269
- VernacularTitle:孤立性皮质下梗死病灶位置与早期神经功能恶化的相关性研究
- Author:
Qian LI
1
;
Chaoping YANG
1
Author Information
1. Cangzhou Central Hospital,Cangzhou 061000,China
- Publication Type:Journal Article
- Keywords:
Isolated subcortical infarction;
Infarction location;
Early neurological deterioration
- From:
Journal of Apoplexy and Nervous Diseases
2022;39(12):1074-1077
- CountryChina
- Language:Chinese
-
Abstract:
Objective To study the relationship between infarct location and early neurological deterioration(END) in isolated subcortical infarctions.Methods We collected patients who were admitted with a diagnosis of isolated subcortical infarction.According to their infarct location,patients were divided into proximal infarct group and distal infarct group.National Institutes of Health Stroke Scale (NIHSS) score was used to assess the degree of neurological impairment in both groups on the day of admission and within 3 days after admission.If the NIHSS score increased by 2 points or more within 72 hours after admission,END was considered.Baseline data and the incidence of END were compared between groups.Finally,We screened the independent risk factors for early prediction of END.Results A total of 422 cases were collected,68 of 239 cases with proximal infarct and 13 of 183 cases with distal infarct developed END.There were statistically significant differences in age,male,LDL,history of stroke,proximal infarct and carrier artery stenosis in END,compared with non-END.Logistic regression analysis showed that age,male,history of stroke,proximal infarct and carrier artery stenosis were independent risk factors for END.Conclusion Patients with proximal infarct are more likely to develop END in isolated subcortical infarctions,which should arouse clinical attention.
- Full text:2024071721595391034Study on the correlation between the location and early neurological deterioration in isolated subcortical infarction.pdf