Nongoitrous autoimmune thyroiditis with facial palsy.
10.6065/apem.2013.18.4.214
- Author:
Hyung Jik LEE
1
;
Jin Kyung KIM
Author Information
1. Department of Pediatrics, Catholic University of Daegu School of Medicine, Daegu, Korea. kimjk@cu.ac.kr
- Publication Type:Case Report
- Keywords:
Autoimmune thyroiditis;
Facial palsy;
Hypothyroidism
- MeSH:
Adolescent;
Antibodies;
Biological Assay;
Facial Paralysis*;
Humans;
Hyperplasia;
Hypothyroidism;
Magnetic Resonance Imaging;
Male;
Paralysis;
Peroxidase;
Pituitary Gland;
Prednisolone;
Radionuclide Imaging;
Receptors, Thyrotropin;
Thyroid Gland;
Thyroiditis, Autoimmune*;
Thyrotropin;
Thyroxine
- From:Annals of Pediatric Endocrinology & Metabolism
2013;18(4):214-217
- CountryRepublic of Korea
- Language:English
-
Abstract:
We report a case of severe hypothyroidism with nongoitrous, autoimmune thyroiditis and pituitary hyperplasia in a 13-year-old boy, who presented with sudden palsy on the left side of his face. Prednisolone and antiviral medication was administered. However, the facial palsy did not improve completely. The medications were replaced with thyroxine, and the facial palsy recovered. Endocrinological testing showed severe hypothyroidism as follows: thyroid stimulating hormone (TSH) level >100 microIU/mL, T4 of 1.04 microg/dL, T3 of 0.31 ng/mL, and free T4 of 0.07 ng/dL. Level of serum antithyroid peroxidase antibodies was 1,933.39 IU/mL, and that of antithyroglobulin antibodies was 848.16 IU/mL. Level of TSH receptor antibodies was >40 IU/L. Bioassay result for TSH receptor stimulating antibodies was negative. Thyroid sonography revealed no increase in the size or vascularity of the bilateral gland. Thyroid scintigraphy with 99mTc showed decreased uptake, and magnetic resonance imaging demonstrated an enlarged pituitary gland.
