Vestibular Paroxysmia and facial spasm may be caused by vascular compression of the vestibular and facial root entry zone. We report a case of paroxysmal nystagmus accompanied by facial spasm and which is well visualized by three-dimensional reconstruction images. The 3-dimensional reconstruction image supports the view that vestibular paroxysmia may occur with hemifacial spasm simultaneously due to vascular compression.
Hemifacial Spasm ; Spasm

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Hemifacial Spasm*

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Hemifacial Spasm*

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Hemifacial Spasm* ; Parotid Gland*

No abstract available.
Blinking* ; Facial Nerve* ; Hemifacial Spasm*

The result of microvascular decompression(MVD) in 105 consecutive cases is presented. The symptomatic vasculoneural compressions including tumor, vascular malformation, or aneurysm are excluded in this study group. The 41 patients with trigeminal neuralgia(TN) were all treated by posterior fossa exploration(PEE). Eighteen of them were treated with MVD only, another 14 with partial sensory rhizotomy(PSR) only, and in the other 9 patients, MVD and PSR were performed together. Sixty-twp of 64 hemifacial spasm(HFS) cases were treated succesfully with MVD via PFE. However in one case of which the offending vessel was not identified, neurolysis was carried out. The other case who had both TN and HFS, died due to intraoperative cardiac arrest. The result of operation was evaluated at one month after the operation. In TN, among thirty-nine(98%) patients who resulted in pain-improvement, 32(82%) experienced complete resolution of the pain, The rate complete pain-relief was higher in the 'MVD only group' than in 'group with PSR'. Among 62(94%) cases of HFS, 29(47%) patients obtained 'complete cure' and 'improvement-only' respectively ; only 4(6%) patients had no change of the spasm. The preoperative clinical characteristics, intraoperative surgical findings and postoperative outcomes were itemized and analyzed find out the prognostic factors. The our results are compared with others in the literature.
Aneurysm ; Decompression* ; Heart Arrest ; Hemifacial Spasm* ; Humans ; Microvascular Decompression Surgery ; Spasm ; Trigeminal Neuralgia* ; Vascular Malformations

The hemifacial spasm has been proved a state of compression usually by blood vessels to the facial nerve. The vascular compression is usually obvious at operation. Since 1980 we have operated microsurgical decompression of 42 hemifacial spasm. Recently we have experienced a case in which venous running in an anterior posterior direction across the caudal aspect of the root entry zone of the facial nerve caused the spasm was coagulated and divided. After operation the patient improved and he free of facial spasm.
Blood Vessels ; Decompression* ; Facial Nerve ; Hemifacial Spasm* ; Humans ; Running ; Spasm ; Veins

Patients with hemifacial spasm were treated with a radiofrequency stimulating assisted percutaneous radiofrequency facial nerve coagulation around the stylomastoid foramen. The initial series of 18 cases of facial spasm are described. The series included 10 men and 8 women, aged from 19 to 75 years. All patients had classical type intractable persistent hemifacial spasm. Although severe facial weakness was made after the procedure to control the heperactive dysfunction of facial nerve in all patient immediately after making lesion, all of them recovered within 6 months follow-up. The longest follow up has been 26 months and only one patient had repeated procedure who experienced recurrence of hemifacial spasm 6 months after initial procedure. The procedure is simple, easy to perform and has no definite complication except transient facial palsy.
Facial Nerve ; Facial Paralysis ; Female ; Follow-Up Studies ; Hemifacial Spasm* ; Humans ; Male ; Recurrence ; Spasm

Two hundred sixty two patients of the essential blepharospasm and hemifacial spasm were treated with Botulinum toxin A (Oculinum(R)). A total of 620 treatments was given injections over a 3-year period. A reduction in spasm intensity was noted in most patients, and the mean response time of the essential blepharospasm and hemifacial spasm were 144.2 days and 172.3 days, respectively, There was no clear relationship between age, sex, toxin dose or preinjected spasm intensity, the amount of spasm reduction, and the mean response time. The mean respone time had no difference from the first through the fourth treatments; but in hemifacial spasm, the mean response time of the second treatment was longer than that of first, third and fourth treatments. The lagophthalmos and superficial punctate keratitis were the most frequent complications. As a result of the injection to avoid the center of the upper and lower eyelids, the frequency of complications could be minimized. There was no clear difference in the beneficial effect and the mcidence of complication (lagophthalmos) between toxin stored in the vaccum and in the non-vaccum state. Patients who were treated with lidocaine mixed toxin had a less effective result than those with saline-mixed toxin.
Blepharospasm* ; Botulinum Toxins* ; Eyelids ; Hemifacial Spasm* ; Humans ; Keratitis ; Lidocaine ; Reaction Time ; Spasm

Thirty-nine patients with blepharospasm were treated with botulinum A toxin. Twenty-six patients had essential blepharospasm, and thirteen had hemifacial spasm. A total of 113 injections were given, and average follow-up was 14.6 months. The mean preinjection spasm intensity was 2.9+, and the mean post-injection spasm intensity was 0.7+. The mean interval between injections was 4.4 months. The treatment was effective, although transient, in all patients with essential blepharospasm and hemifacial spasm. The toxin had a prolonged effect on the patients who had previously undergone muscle stripping proced ure. The side effects were mild, transient, and local.
Blepharospasm* ; Botulinum Toxins, Type A* ; Follow-Up Studies ; Hemifacial Spasm ; Humans ; Spasm